Nitric oxide synthase in cerebral ischemia. Possible contribution of nitric oxide synthase activation in brain microvessels to cerebral ischemic injury

The results of our continuing studies on the role of nitric oxide (NO) in cellular mechanisms of ischemic brain damage as well as related reports from other laboratories are summarized in this paper. Repetitive ip administration of NG-nitro-L-arginine (L-NNA), a NO synthase (NOS) inhibitor, protecte...

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Veröffentlicht in:	Molecular and chemical neuropathology 1995-10, Vol.26 (2), p.107-157
Hauptverfasser:	Nagafuji, T, Sugiyama, M, Matsui, T, Muto, A, Naito, S
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Brain - blood supply Brain - drug effects Brain - enzymology Enzyme Activation Enzyme Inhibitors - pharmacology Ischemic Attack, Transient - enzymology Ischemic Attack, Transient - pathology Ischemic Attack, Transient - physiopathology Microcirculation - drug effects Nitric Oxide - biosynthesis Nitric Oxide - physiology Nitric Oxide Synthase - drug effects Prosencephalon - drug effects Rats
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