1,3-Dipropyl-8-cyclopentylxanthine attenuates the NMDA response to hypoxia in the rat hippocampus

Excitatory amino acids may cause neuronal damage and death in cerebral hypoxia and ischemia, through the activation of different subtypes of glutamate receptors, in particular of the N- methyl- d- aspartate (NMDA) receptor. In the present work, the effect of hypoxia on the component of the field exc...

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Veröffentlicht in:	Brain research 1994-10, Vol.661 (1), p.265-273
Hauptverfasser:	Canhão, Patrícia, Mendonça, Alexandre de, Ribeiro, J.Alexandre
Format:	Artikel
Sprache:	eng
Schlagworte:	1,3-Dipropyl-8-cyclopentylxanthine Adenosine Animals Biological and medical sciences Evoked Potentials - drug effects Excitatory postsynaptic potential Glutamate Hippocampus Hippocampus - drug effects Hippocampus - physiology Hypoxia In Vitro Techniques Male Medical sciences N-Methyl- d-aspartate N-Methylaspartate - pharmacology Neuropharmacology Neuroprotective agent Pharmacology. Drug treatments Purinergic P1 Receptor Antagonists Pyramidal Cells - drug effects Pyramidal Cells - physiology Rats Rats, Wistar Receptors, N-Methyl-D-Aspartate - physiology Synaptic Transmission - drug effects Time Factors Xanthines - pharmacology
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