Four Chamber Pacing in Dilated Cardiomyopathy
A 54‐year‐old man received a four chamber pacing system for severe congestive heart failure (NYHA functional Class IV). His ECG showed a left bundle branch block (200‐msec QHS duration) with 200‐msec PR interval, normal QRS axis, and 90‐msec interatrial interval. An acute hemodynamic study with inse...
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Veröffentlicht in: | Pacing and clinical electrophysiology 1994-11, Vol.17 (11), p.1974-1979 |
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container_end_page | 1979 |
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container_issue | 11 |
container_start_page | 1974 |
container_title | Pacing and clinical electrophysiology |
container_volume | 17 |
creator | CAZEAU, S. RITTER, P. BAKDACH, S. LAZARUS, A. LIMOUSIN, M. HENAO, L. MUNDLER, O. DAUBERT, J.C. MUGICA, J. |
description | A 54‐year‐old man received a four chamber pacing system for severe congestive heart failure (NYHA functional Class IV). His ECG showed a left bundle branch block (200‐msec QHS duration) with 200‐msec PR interval, normal QRS axis, and 90‐msec interatrial interval. An acute hemodynamic study with insertion of four temporary leads was performed prior to the implant, which demonstrated a significant increase in cardiac output and decrease of pulmonary capillary wedge pressure. A permanent pacemaker was implanted based on the encouraging results of the acute study. The right chamber leads were introduced by cephalic and suhcla vian approaches. The left atrium was paced with a coronary sinus lead, Medtronic SP 2188–58 model. An epicardial Medtronic 5071 lead was placed on the LV free wall. The four leads were connected to a standard bipolar DDD pacemaker. Chorus 6234. The two atrial leads were connected via a Y‐connector to the atrial channel of the pacemaker with a bipolar pacing configuration. The two ventricular leads were connected in a similar fashion to the ventricular channel of the device. The right chamber leads were connected to the distal poles. The left chamber leads were connected to the proximal poles of the pacemaker. Six weeks later, the patient's clinical status improved markedly with a weight loss of 17 kg and disappearance of peripheral edema. His functional class was reduced to NYHA II. Four chamber pacing is technically feasible. In patients with evidence of interventricular dyssynchrony, this original pacing mode probably provides a mechanical activation sequence closer to the natural one. We doubt that this technique will have an impact on long‐term survival, but it could be of major importance to improve the patient's well‐being and control heart failure. |
doi_str_mv | 10.1111/j.1540-8159.1994.tb03783.x |
format | Article |
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His ECG showed a left bundle branch block (200‐msec QHS duration) with 200‐msec PR interval, normal QRS axis, and 90‐msec interatrial interval. An acute hemodynamic study with insertion of four temporary leads was performed prior to the implant, which demonstrated a significant increase in cardiac output and decrease of pulmonary capillary wedge pressure. A permanent pacemaker was implanted based on the encouraging results of the acute study. The right chamber leads were introduced by cephalic and suhcla vian approaches. The left atrium was paced with a coronary sinus lead, Medtronic SP 2188–58 model. An epicardial Medtronic 5071 lead was placed on the LV free wall. The four leads were connected to a standard bipolar DDD pacemaker. Chorus 6234. The two atrial leads were connected via a Y‐connector to the atrial channel of the pacemaker with a bipolar pacing configuration. The two ventricular leads were connected in a similar fashion to the ventricular channel of the device. The right chamber leads were connected to the distal poles. The left chamber leads were connected to the proximal poles of the pacemaker. Six weeks later, the patient's clinical status improved markedly with a weight loss of 17 kg and disappearance of peripheral edema. His functional class was reduced to NYHA II. Four chamber pacing is technically feasible. In patients with evidence of interventricular dyssynchrony, this original pacing mode probably provides a mechanical activation sequence closer to the natural one. We doubt that this technique will have an impact on long‐term survival, but it could be of major importance to improve the patient's well‐being and control heart failure.</description><identifier>ISSN: 0147-8389</identifier><identifier>EISSN: 1540-8159</identifier><identifier>DOI: 10.1111/j.1540-8159.1994.tb03783.x</identifier><identifier>PMID: 7845801</identifier><language>eng</language><publisher>Oxford, UK: Blackwell Publishing Ltd</publisher><subject>Bundle-Branch Block - etiology ; Bundle-Branch Block - therapy ; Cardiac Output ; Cardiac Pacing, Artificial ; Cardiomyopathy, Dilated - complications ; Cardiomyopathy, Dilated - physiopathology ; Cardiomyopathy, Dilated - therapy ; dilated cardiomyopathy ; Electrocardiography ; four chamber pacing ; hemodynamics ; Humans ; Male ; Middle Aged ; Pacemaker, Artificial ; Pulmonary Wedge Pressure</subject><ispartof>Pacing and clinical electrophysiology, 1994-11, Vol.17 (11), p.1974-1979</ispartof><lds50>peer_reviewed</lds50><woscitedreferencessubscribed>false</woscitedreferencessubscribed><citedby>FETCH-LOGICAL-c3904-f259d454a6188b06757769f18d3b40c41b010f471691f280fd8600d44471f9183</citedby><cites>FETCH-LOGICAL-c3904-f259d454a6188b06757769f18d3b40c41b010f471691f280fd8600d44471f9183</cites></display><links><openurl>$$Topenurl_article</openurl><openurlfulltext>$$Topenurlfull_article</openurlfulltext><thumbnail>$$Tsyndetics_thumb_exl</thumbnail><linktopdf>$$Uhttps://onlinelibrary.wiley.com/doi/pdf/10.1111%2Fj.1540-8159.1994.tb03783.x$$EPDF$$P50$$Gwiley$$H</linktopdf><linktohtml>$$Uhttps://onlinelibrary.wiley.com/doi/full/10.1111%2Fj.1540-8159.1994.tb03783.x$$EHTML$$P50$$Gwiley$$H</linktohtml><link.rule.ids>314,776,780,1411,27901,27902,45550,45551</link.rule.ids><backlink>$$Uhttps://www.ncbi.nlm.nih.gov/pubmed/7845801$$D View this record in MEDLINE/PubMed$$Hfree_for_read</backlink></links><search><creatorcontrib>CAZEAU, S.</creatorcontrib><creatorcontrib>RITTER, P.</creatorcontrib><creatorcontrib>BAKDACH, S.</creatorcontrib><creatorcontrib>LAZARUS, A.</creatorcontrib><creatorcontrib>LIMOUSIN, M.</creatorcontrib><creatorcontrib>HENAO, L.</creatorcontrib><creatorcontrib>MUNDLER, O.</creatorcontrib><creatorcontrib>DAUBERT, J.C.</creatorcontrib><creatorcontrib>MUGICA, J.</creatorcontrib><title>Four Chamber Pacing in Dilated Cardiomyopathy</title><title>Pacing and clinical electrophysiology</title><addtitle>Pacing Clin Electrophysiol</addtitle><description>A 54‐year‐old man received a four chamber pacing system for severe congestive heart failure (NYHA functional Class IV). His ECG showed a left bundle branch block (200‐msec QHS duration) with 200‐msec PR interval, normal QRS axis, and 90‐msec interatrial interval. An acute hemodynamic study with insertion of four temporary leads was performed prior to the implant, which demonstrated a significant increase in cardiac output and decrease of pulmonary capillary wedge pressure. A permanent pacemaker was implanted based on the encouraging results of the acute study. The right chamber leads were introduced by cephalic and suhcla vian approaches. The left atrium was paced with a coronary sinus lead, Medtronic SP 2188–58 model. An epicardial Medtronic 5071 lead was placed on the LV free wall. The four leads were connected to a standard bipolar DDD pacemaker. Chorus 6234. The two atrial leads were connected via a Y‐connector to the atrial channel of the pacemaker with a bipolar pacing configuration. The two ventricular leads were connected in a similar fashion to the ventricular channel of the device. The right chamber leads were connected to the distal poles. The left chamber leads were connected to the proximal poles of the pacemaker. Six weeks later, the patient's clinical status improved markedly with a weight loss of 17 kg and disappearance of peripheral edema. His functional class was reduced to NYHA II. Four chamber pacing is technically feasible. In patients with evidence of interventricular dyssynchrony, this original pacing mode probably provides a mechanical activation sequence closer to the natural one. We doubt that this technique will have an impact on long‐term survival, but it could be of major importance to improve the patient's well‐being and control heart failure.</description><subject>Bundle-Branch Block - etiology</subject><subject>Bundle-Branch Block - therapy</subject><subject>Cardiac Output</subject><subject>Cardiac Pacing, Artificial</subject><subject>Cardiomyopathy, Dilated - complications</subject><subject>Cardiomyopathy, Dilated - physiopathology</subject><subject>Cardiomyopathy, Dilated - therapy</subject><subject>dilated cardiomyopathy</subject><subject>Electrocardiography</subject><subject>four chamber pacing</subject><subject>hemodynamics</subject><subject>Humans</subject><subject>Male</subject><subject>Middle Aged</subject><subject>Pacemaker, Artificial</subject><subject>Pulmonary Wedge Pressure</subject><issn>0147-8389</issn><issn>1540-8159</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>1994</creationdate><recordtype>article</recordtype><sourceid>EIF</sourceid><recordid>eNqVkE1PwzAMhiMEgjH4CUgVB24t9pI0CSegfErTQAIEtyhtU-ho15F0Yvv3tNq0O75Y8ms_lh5CThEi7Op8GiFnEErkKkKlWNSmQIWk0XKHDLbRLhkAMhFKKtUBOfR-CgAxML5P9oVkXAIOSHjXLFyQfJk6tS54Nlk5-wzKWXBTVqa1eZAYl5dNvWrmpv1aHZG9wlTeHm_6kLzd3b4mD-H46f4xuRqHGVXAwmLEVc44MzFKmUIsuBCxKlDmNGWQMUwBoWACY4XFSEKRyxggZ6wbFQolHZKzNXfump-F9a2uS5_ZqjIz2yy8FkIgCOgXL9aLmWu8d7bQc1fWxq00gu5d6anuheheiO5d6Y0rveyOTzZfFmlt8-3pRk6XX67z37Kyq3-Q9fNVcotKsA4RrhGlb-1yizDuW8eCCq7fJ_f6ejL-4KMX0JT-AZsGha4</recordid><startdate>199411</startdate><enddate>199411</enddate><creator>CAZEAU, S.</creator><creator>RITTER, P.</creator><creator>BAKDACH, S.</creator><creator>LAZARUS, A.</creator><creator>LIMOUSIN, M.</creator><creator>HENAO, L.</creator><creator>MUNDLER, O.</creator><creator>DAUBERT, J.C.</creator><creator>MUGICA, J.</creator><general>Blackwell Publishing Ltd</general><scope>BSCLL</scope><scope>CGR</scope><scope>CUY</scope><scope>CVF</scope><scope>ECM</scope><scope>EIF</scope><scope>NPM</scope><scope>AAYXX</scope><scope>CITATION</scope><scope>7X8</scope></search><sort><creationdate>199411</creationdate><title>Four Chamber Pacing in Dilated Cardiomyopathy</title><author>CAZEAU, S. ; RITTER, P. ; BAKDACH, S. ; LAZARUS, A. ; LIMOUSIN, M. ; HENAO, L. ; MUNDLER, O. ; DAUBERT, J.C. ; MUGICA, J.</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-c3904-f259d454a6188b06757769f18d3b40c41b010f471691f280fd8600d44471f9183</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng</language><creationdate>1994</creationdate><topic>Bundle-Branch Block - etiology</topic><topic>Bundle-Branch Block - therapy</topic><topic>Cardiac Output</topic><topic>Cardiac Pacing, Artificial</topic><topic>Cardiomyopathy, Dilated - complications</topic><topic>Cardiomyopathy, Dilated - physiopathology</topic><topic>Cardiomyopathy, Dilated - therapy</topic><topic>dilated cardiomyopathy</topic><topic>Electrocardiography</topic><topic>four chamber pacing</topic><topic>hemodynamics</topic><topic>Humans</topic><topic>Male</topic><topic>Middle Aged</topic><topic>Pacemaker, Artificial</topic><topic>Pulmonary Wedge Pressure</topic><toplevel>peer_reviewed</toplevel><toplevel>online_resources</toplevel><creatorcontrib>CAZEAU, S.</creatorcontrib><creatorcontrib>RITTER, P.</creatorcontrib><creatorcontrib>BAKDACH, S.</creatorcontrib><creatorcontrib>LAZARUS, A.</creatorcontrib><creatorcontrib>LIMOUSIN, M.</creatorcontrib><creatorcontrib>HENAO, L.</creatorcontrib><creatorcontrib>MUNDLER, O.</creatorcontrib><creatorcontrib>DAUBERT, J.C.</creatorcontrib><creatorcontrib>MUGICA, J.</creatorcontrib><collection>Istex</collection><collection>Medline</collection><collection>MEDLINE</collection><collection>MEDLINE (Ovid)</collection><collection>MEDLINE</collection><collection>MEDLINE</collection><collection>PubMed</collection><collection>CrossRef</collection><collection>MEDLINE - Academic</collection><jtitle>Pacing and clinical electrophysiology</jtitle></facets><delivery><delcategory>Remote Search Resource</delcategory><fulltext>fulltext</fulltext></delivery><addata><au>CAZEAU, S.</au><au>RITTER, P.</au><au>BAKDACH, S.</au><au>LAZARUS, A.</au><au>LIMOUSIN, M.</au><au>HENAO, L.</au><au>MUNDLER, O.</au><au>DAUBERT, J.C.</au><au>MUGICA, J.</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>Four Chamber Pacing in Dilated Cardiomyopathy</atitle><jtitle>Pacing and clinical electrophysiology</jtitle><addtitle>Pacing Clin Electrophysiol</addtitle><date>1994-11</date><risdate>1994</risdate><volume>17</volume><issue>11</issue><spage>1974</spage><epage>1979</epage><pages>1974-1979</pages><issn>0147-8389</issn><eissn>1540-8159</eissn><abstract>A 54‐year‐old man received a four chamber pacing system for severe congestive heart failure (NYHA functional Class IV). His ECG showed a left bundle branch block (200‐msec QHS duration) with 200‐msec PR interval, normal QRS axis, and 90‐msec interatrial interval. An acute hemodynamic study with insertion of four temporary leads was performed prior to the implant, which demonstrated a significant increase in cardiac output and decrease of pulmonary capillary wedge pressure. A permanent pacemaker was implanted based on the encouraging results of the acute study. The right chamber leads were introduced by cephalic and suhcla vian approaches. The left atrium was paced with a coronary sinus lead, Medtronic SP 2188–58 model. An epicardial Medtronic 5071 lead was placed on the LV free wall. The four leads were connected to a standard bipolar DDD pacemaker. Chorus 6234. The two atrial leads were connected via a Y‐connector to the atrial channel of the pacemaker with a bipolar pacing configuration. The two ventricular leads were connected in a similar fashion to the ventricular channel of the device. The right chamber leads were connected to the distal poles. The left chamber leads were connected to the proximal poles of the pacemaker. Six weeks later, the patient's clinical status improved markedly with a weight loss of 17 kg and disappearance of peripheral edema. His functional class was reduced to NYHA II. Four chamber pacing is technically feasible. In patients with evidence of interventricular dyssynchrony, this original pacing mode probably provides a mechanical activation sequence closer to the natural one. We doubt that this technique will have an impact on long‐term survival, but it could be of major importance to improve the patient's well‐being and control heart failure.</abstract><cop>Oxford, UK</cop><pub>Blackwell Publishing Ltd</pub><pmid>7845801</pmid><doi>10.1111/j.1540-8159.1994.tb03783.x</doi><tpages>6</tpages></addata></record> |
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subjects | Bundle-Branch Block - etiology Bundle-Branch Block - therapy Cardiac Output Cardiac Pacing, Artificial Cardiomyopathy, Dilated - complications Cardiomyopathy, Dilated - physiopathology Cardiomyopathy, Dilated - therapy dilated cardiomyopathy Electrocardiography four chamber pacing hemodynamics Humans Male Middle Aged Pacemaker, Artificial Pulmonary Wedge Pressure |
title | Four Chamber Pacing in Dilated Cardiomyopathy |
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