Chronological changes of arterial diameter, cGMP, and protein kinase C in the development of vasospasm

We hypothesized that nitric oxide exerts a negative feedback control on protein kinase C (PKC) activation, and the disturbance of the feedback control after subarachnoid hemorrhage results in vasospasm due to PKC activation. This study was undertaken to verify this hypothesis. Different dogs were pr...

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Veröffentlicht in:	Stroke (1970) 1995-10, Vol.26 (10), p.1916-1921
Hauptverfasser:	Nishizawa, S, Yamamoto, S, Yokoyama, T, Ryu, H, Uemura, K
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Basilar Artery - enzymology Basilar Artery - pathology Cerebellum - blood supply Cerebellum - enzymology Cerebellum - pathology Cerebral Angiography Cyclic GMP - metabolism Disease Models, Animal Dogs Enzyme Activation Feedback Female Ischemic Attack, Transient - enzymology Ischemic Attack, Transient - etiology Ischemic Attack, Transient - pathology Male Muscle, Smooth, Vascular - enzymology Muscle, Smooth, Vascular - pathology Nitric Oxide - physiology Protein Kinase C - metabolism Subarachnoid Hemorrhage - enzymology Subarachnoid Hemorrhage - pathology Time Factors
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