Catabolic effects of thyroid hormone excess: The contribution of adrenergic activity to hypermetabolism and protein breakdown

Although patients with thyrotoxicosis improve clinically after treatment with beta-adrenergic blocking drugs, it has never been established whether the hypermetabolism and body protein wasting caused by thyroid hormone excess are actually mediated by adrenergic mechanisms. To evaluate this issue, we...

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Veröffentlicht in:	Metabolism, clinical and experimental clinical and experimental, 1987-06, Vol.36 (6), p.562-569
Hauptverfasser:	Gelfand, Robert A., Hutchinson-Williams, Karen A., Bonde, Alfons A., Castellino, Pietro, Sherwin, Robert S.
Format:	Artikel
Sprache:	eng
Schlagworte:	Adrenergic beta-Antagonists - therapeutic use Adrenergic Fibers - physiopathology Adult Biological and medical sciences Body Weight Endocrinopathies Energy Intake Energy Metabolism Epinephrine - pharmacology Female Humans Kinetics Leucine - metabolism Male Medical sciences Nitrogen - metabolism Non tumoral diseases. Target tissue resistance. Benign neoplasms Propranolol - therapeutic use Proteins - metabolism Thyroid. Thyroid axis (diseases) Thyrotoxicosis - drug therapy Thyrotoxicosis - metabolism Thyrotoxicosis - physiopathology Triiodothyronine - pharmacology
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