Oligohydramnios sequence (Potter's syndrome): case clustering in northeastern Tennessee
The oligohydramnios sequence (OS) is manifest in newborns when prolonged oligohydramnios has been present during pregnancy. The most important signs are an infant small for gestational age, with wrinkled skin, Potter facies, compression deformities of the limbs, and respiratory distress caused by pu...
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| Veröffentlicht in: | Southern medical journal (Birmingham, Ala.) Ala.), 1987-05, Vol.80 (5), p.585-592 |
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| container_title | Southern medical journal (Birmingham, Ala.) |
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| creator | Prouty, L A Myers, T L |
| description | The oligohydramnios sequence (OS) is manifest in newborns when prolonged oligohydramnios has been present during pregnancy. The most important signs are an infant small for gestational age, with wrinkled skin, Potter facies, compression deformities of the limbs, and respiratory distress caused by pulmonary hypoplasia. The recurrence rate of kidney conditions implicated in OS depends on the severity of the condition in the previously affected sib. While absent or encysted kidneys (Potter types II and IV) are incompatible with life, chronic leakage of amniotic fluid may result in a viable infant who shows some signs of OS. The usual incidence of absent or encysted kidneys is 1 per 6,250 births. This rate was exceeded recently in northeastern Tennessee, in a clustering of cases for which no environmental causes are evident. |
| doi_str_mv | 10.1097/00007611-198705000-00010 |
| format | Article |
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The most important signs are an infant small for gestational age, with wrinkled skin, Potter facies, compression deformities of the limbs, and respiratory distress caused by pulmonary hypoplasia. The recurrence rate of kidney conditions implicated in OS depends on the severity of the condition in the previously affected sib. While absent or encysted kidneys (Potter types II and IV) are incompatible with life, chronic leakage of amniotic fluid may result in a viable infant who shows some signs of OS. The usual incidence of absent or encysted kidneys is 1 per 6,250 births. This rate was exceeded recently in northeastern Tennessee, in a clustering of cases for which no environmental causes are evident.</description><identifier>ISSN: 0038-4348</identifier><identifier>DOI: 10.1097/00007611-198705000-00010</identifier><identifier>PMID: 3576270</identifier><language>eng</language><publisher>United States</publisher><subject>Abnormalities, Multiple - epidemiology ; Abnormalities, Multiple - etiology ; Amniotic Fluid - secretion ; Facial Expression ; Female ; Humans ; Infant, Newborn ; Infant, Small for Gestational Age ; Karyotyping ; Kidney - abnormalities ; Polycystic Kidney Diseases - complications ; Probability ; Recurrence ; Sex Factors ; Syndrome ; Tennessee</subject><ispartof>Southern medical journal (Birmingham, Ala.), 1987-05, Vol.80 (5), p.585-592</ispartof><lds50>peer_reviewed</lds50><woscitedreferencessubscribed>false</woscitedreferencessubscribed></display><links><openurl>$$Topenurl_article</openurl><openurlfulltext>$$Topenurlfull_article</openurlfulltext><thumbnail>$$Tsyndetics_thumb_exl</thumbnail><link.rule.ids>314,776,780,27901,27902</link.rule.ids><backlink>$$Uhttps://www.ncbi.nlm.nih.gov/pubmed/3576270$$D View this record in MEDLINE/PubMed$$Hfree_for_read</backlink></links><search><creatorcontrib>Prouty, L A</creatorcontrib><creatorcontrib>Myers, T L</creatorcontrib><title>Oligohydramnios sequence (Potter's syndrome): case clustering in northeastern Tennessee</title><title>Southern medical journal (Birmingham, Ala.)</title><addtitle>South Med J</addtitle><description>The oligohydramnios sequence (OS) is manifest in newborns when prolonged oligohydramnios has been present during pregnancy. The most important signs are an infant small for gestational age, with wrinkled skin, Potter facies, compression deformities of the limbs, and respiratory distress caused by pulmonary hypoplasia. The recurrence rate of kidney conditions implicated in OS depends on the severity of the condition in the previously affected sib. While absent or encysted kidneys (Potter types II and IV) are incompatible with life, chronic leakage of amniotic fluid may result in a viable infant who shows some signs of OS. The usual incidence of absent or encysted kidneys is 1 per 6,250 births. This rate was exceeded recently in northeastern Tennessee, in a clustering of cases for which no environmental causes are evident.</description><subject>Abnormalities, Multiple - epidemiology</subject><subject>Abnormalities, Multiple - etiology</subject><subject>Amniotic Fluid - secretion</subject><subject>Facial Expression</subject><subject>Female</subject><subject>Humans</subject><subject>Infant, Newborn</subject><subject>Infant, Small for Gestational Age</subject><subject>Karyotyping</subject><subject>Kidney - abnormalities</subject><subject>Polycystic Kidney Diseases - complications</subject><subject>Probability</subject><subject>Recurrence</subject><subject>Sex Factors</subject><subject>Syndrome</subject><subject>Tennessee</subject><issn>0038-4348</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>1987</creationdate><recordtype>article</recordtype><sourceid>EIF</sourceid><recordid>eNo9kMlOwzAQhn0AlVJ4BCSfWA4Bu46XcEMVm1SpHIo4Wo49aYMSp9jJoW-PS0tHGo1m5p9FH0KYkntKCvlAkklBaUYLJQlPWZackhM0JoSpLGe5OkPnMX7vhFyJERoxLsVUkjH6WjT1qltvXTCtr7uII_wM4C3g24-u7yHcpNLWu9C1cPeIrYmAbTPE1Kn9Ctce-y70azC7isdL8B5iBLhAp5VpIlwe4gR9vjwvZ2_ZfPH6PnuaZ3bKeZ9VyhROToUpLTBLOVcAEgopgBJaiNwxUZXCTZUTtqB5aSvFy8JRBkaKXHA2Qdf7vZvQpcdjr9s6Wmga46EbopaSU6YKmYRqL7ShizFApTehbk3Yakr0jqP-56iPHPUfxzR6dbgxlC244-ABIvsF6KNxKA</recordid><startdate>19870501</startdate><enddate>19870501</enddate><creator>Prouty, L A</creator><creator>Myers, T L</creator><scope>CGR</scope><scope>CUY</scope><scope>CVF</scope><scope>ECM</scope><scope>EIF</scope><scope>NPM</scope><scope>AAYXX</scope><scope>CITATION</scope><scope>7X8</scope></search><sort><creationdate>19870501</creationdate><title>Oligohydramnios sequence (Potter's syndrome): case clustering in northeastern Tennessee</title><author>Prouty, L A ; Myers, T L</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-c255t-f8a9d726abce3c1558ee7e976e101964d36fb6d28d6c914bcf85b9d13ea764653</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng</language><creationdate>1987</creationdate><topic>Abnormalities, Multiple - epidemiology</topic><topic>Abnormalities, Multiple - etiology</topic><topic>Amniotic Fluid - secretion</topic><topic>Facial Expression</topic><topic>Female</topic><topic>Humans</topic><topic>Infant, Newborn</topic><topic>Infant, Small for Gestational Age</topic><topic>Karyotyping</topic><topic>Kidney - abnormalities</topic><topic>Polycystic Kidney Diseases - complications</topic><topic>Probability</topic><topic>Recurrence</topic><topic>Sex Factors</topic><topic>Syndrome</topic><topic>Tennessee</topic><toplevel>peer_reviewed</toplevel><toplevel>online_resources</toplevel><creatorcontrib>Prouty, L A</creatorcontrib><creatorcontrib>Myers, T L</creatorcontrib><collection>Medline</collection><collection>MEDLINE</collection><collection>MEDLINE (Ovid)</collection><collection>MEDLINE</collection><collection>MEDLINE</collection><collection>PubMed</collection><collection>CrossRef</collection><collection>MEDLINE - Academic</collection><jtitle>Southern medical journal (Birmingham, Ala.)</jtitle></facets><delivery><delcategory>Remote Search Resource</delcategory><fulltext>fulltext</fulltext></delivery><addata><au>Prouty, L A</au><au>Myers, T L</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>Oligohydramnios sequence (Potter's syndrome): case clustering in northeastern Tennessee</atitle><jtitle>Southern medical journal (Birmingham, Ala.)</jtitle><addtitle>South Med J</addtitle><date>1987-05-01</date><risdate>1987</risdate><volume>80</volume><issue>5</issue><spage>585</spage><epage>592</epage><pages>585-592</pages><issn>0038-4348</issn><abstract>The oligohydramnios sequence (OS) is manifest in newborns when prolonged oligohydramnios has been present during pregnancy. The most important signs are an infant small for gestational age, with wrinkled skin, Potter facies, compression deformities of the limbs, and respiratory distress caused by pulmonary hypoplasia. The recurrence rate of kidney conditions implicated in OS depends on the severity of the condition in the previously affected sib. While absent or encysted kidneys (Potter types II and IV) are incompatible with life, chronic leakage of amniotic fluid may result in a viable infant who shows some signs of OS. The usual incidence of absent or encysted kidneys is 1 per 6,250 births. This rate was exceeded recently in northeastern Tennessee, in a clustering of cases for which no environmental causes are evident.</abstract><cop>United States</cop><pmid>3576270</pmid><doi>10.1097/00007611-198705000-00010</doi><tpages>8</tpages></addata></record> |
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| identifier | ISSN: 0038-4348 |
| ispartof | Southern medical journal (Birmingham, Ala.), 1987-05, Vol.80 (5), p.585-592 |
| issn | 0038-4348 |
| language | eng |
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| source | Journals@Ovid Complete - AutoHoldings; MEDLINE |
| subjects | Abnormalities, Multiple - epidemiology Abnormalities, Multiple - etiology Amniotic Fluid - secretion Facial Expression Female Humans Infant, Newborn Infant, Small for Gestational Age Karyotyping Kidney - abnormalities Polycystic Kidney Diseases - complications Probability Recurrence Sex Factors Syndrome Tennessee |
| title | Oligohydramnios sequence (Potter's syndrome): case clustering in northeastern Tennessee |
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