Increased Oxidative Stress in Experimental Renovascular Hypertension

The pathophysiological mechanisms responsible for maintenance of chronic renovascular hypertension remain undefined. Excess angiotensin II generation may lead to release of reactive oxygen species and increased vasoconstrictor activity. To examine the potential involvement of oxidation-sensitive mec...

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Veröffentlicht in:	Hypertension (Dallas, Tex. 1979) Tex. 1979), 2001-02, Vol.37 (2, Part 2 Suppl), p.541-546
Hauptverfasser:	Lerman, Lilach O, Nath, Karl A, Rodriguez-Porcel, Martin, Krier, James D, Schwartz, Robert S, Napoli, Claudio, Romero, J Carlos
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Arterial hypertension. Arterial hypotension Biological and medical sciences Blood and lymphatic vessels Blood Pressure Cardiology. Vascular system Dinoprost - analogs & derivatives Dinoprost - blood Experimental diseases F2-Isoprostanes Female Free Radical Scavengers - analysis Hypertension, Renovascular - blood Hypertension, Renovascular - etiology Hypertension, Renovascular - physiopathology Kidney Cortex - blood supply Kidney Cortex - metabolism Medical sciences Oxidative Stress Renal Artery Obstruction - complications Renal Circulation Renin - blood Swine Thiobarbituric Acid Reactive Substances - analysis
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