Dilatation of cerebral arterioles in response to N-methyl- d-aspartate : role of CGRP and acetylcholine

The purpose of these experiments was to examine mechanisms by which N-methyl- d-aspartate (NMDA) produces nitric oxide-dependent vasodilatation in brain. Some nitrovasodilators appear to dilate cerebral arterioles, in part, by release of calcitonin gene-related peptide (CGRP) from trigeminal fibers....

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Veröffentlicht in:	Brain research 1994-03, Vol.640 (1), p.93-97
Hauptverfasser:	Faraci, Frank M., Breese, Keith R.
Format:	Artikel
Sprache:	eng
Schlagworte:	Acetylcholine - antagonists & inhibitors Acetylcholine - pharmacology Acetylcholine - physiology Animals Arterioles - drug effects Atropine Atropine - pharmacology Biological and medical sciences Calcitonin Gene-Related Peptide - antagonists & inhibitors Calcitonin Gene-Related Peptide - pharmacology Calcitonin Gene-Related Peptide - physiology Cerebral arteriole Cerebral circulation. Blood-brain barrier. Choroid plexus. Cerebrospinal fluid. Circumventricular organ. Meninges Cerebrovascular Circulation - drug effects Cerebrovascular Circulation - physiology CGRP(8–37) Fundamental and applied biological sciences. Psychology N-Methylaspartate - antagonists & inhibitors N-Methylaspartate - pharmacology Nitric oxide Nitric Oxide - physiology Nitroglycerin Nitroglycerin - pharmacology NMDA Peptide Fragments - pharmacology Rabbits Receptors, Calcitonin Gene-Related Peptide - drug effects Vasodilation - drug effects Vasodilation - physiology Vertebrates: nervous system and sense organs
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