H2O2 alters rat cardiac sarcomere function and protein phosphorylation through redox signaling

ROS, such as H2O2, are a component of pathological conditions in many organ systems and have been reported to be elevated in cardiac pathophysiology. The experiments presented here test the hypothesis that H2O2 induces alterations in cardiac myofilament function by the posttranslational modification...

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Veröffentlicht in:	American Journal of Physiology: Cell Physiology 2010-01, Vol.299 (3), p.H723-H730
Hauptverfasser:	Avner, Benjamin S, Hinken, Aaron C, Yuan, Chao, Solaro, RJohn
Format:	Artikel
Sprache:	eng
Online-Zugang:	Volltext
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