Facilitatory modulation of mesolimbic dopamine neuronal activity by a μ-opioid agonist and nicotine as examined with in vivo microdialysis

Administration of either Tyr- d-Ala-Gly-MePhe-Gly(ol) (DAGO), a μ-opioid agonist, or nicotine into the VTA (A10) caused an increase in both dopamine (DA) and its metabolite levels in both the A10 region and DA nerve terminals in the nucleus accumbens of rats when assessed with dual probe brain dialy...

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Veröffentlicht in:	Brain research 1993-10, Vol.624 (1), p.277-280
Hauptverfasser:	Yoshida, Masami, Yokoo, Hideyasu, Tanaka, Takahiko, Mizoguchi, Katsuhiro, Emoto, Hiroyuki, Ishii, Hideo, Tanaka, Masatoshi
Format:	Artikel
Sprache:	eng
Schlagworte:	3,4-Dihydroxyphenylacetic Acid - metabolism Animals Biological and medical sciences Central nervous system Central neurotransmission. Neuromudulation. Pathways and receptors Dopamine - metabolism Enkephalin, Ala-MePhe-Gly Enkephalins - pharmacology Fundamental and applied biological sciences. Psychology In vivo brain dialysis Limbic System - cytology Limbic System - metabolism Male Mesolimbic dopamine neuron Microdialysis Neurons - metabolism Nicotine Nicotine - pharmacology Opioid Rats Rats, Wistar Receptors, Opioid, mu - physiology Vertebrates: nervous system and sense organs
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