Hypothalamic neurotransmitter function in experimentally induced hyperprolactinemia

It is known that animals or patients bearing a prolactin (PRL)-secreting tumor (PST) do not suppress PRL levels after administration of indirectly acting dopamine agonists, namely nomifensine (Nom), and are not responsive to the PRL releasing effect of antidopaminergic drugs and opioid peptides. Sin...

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Veröffentlicht in:	Brain research 1984-12, Vol.324 (2), p.233-241
Hauptverfasser:	Pen˜alva, Angela, Novelli, Antonello, Parenti, Marco, Locatelli, Vittorio, Mu¨ller, Eugenio E., Cocchi, Daniela
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Biological and medical sciences D-Ala,MePhe,Met-ol-enkephalin - pharmacology Domperidone - pharmacology Dopamine - analysis Dopamine - physiology Endocrinopathies Female hyperprolactinemia hyperprolactinemic rats Hypothalamus - physiology Hypothalamus. Hypophysis. Epiphysis (diseases) Median Eminence - analysis Medical sciences Naloxone - pharmacology Nomifensine - pharmacology Non tumoral diseases. Target tissue resistance. Benign neoplasms opiates opioid receptors opioids peptides prolactin Prolactin - blood Rats Rats, Inbred Strains receptors Synaptic Transmission tuberoinfundibular dopaminergic systm tuberoinfundibular tract
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