Upregulation of GLUT1 expression is necessary for hypertrophy and survival of neonatal rat cardiomyocytes

During hypertrophy the heart increases its utilization of glucose and decreases that of fatty acids, resuming a fetal pattern of substrate metabolism. As demonstrated here, GLUT1 protein expression is increased in association with in vivo pressure-overload-induced hypertrophy. The relationship of ch...

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Veröffentlicht in:	Journal of molecular and cellular cardiology 2003-10, Vol.35 (10), p.1217-1227
Hauptverfasser:	Morissette, Michael R., Howes, Amy L., Zhang, Tong, Heller Brown, Joan
Format:	Artikel
Sprache:	eng
Schlagworte:	Adenoviridae - genetics Animals Aorta - pathology Apoptosis Atrial Natriuretic Factor - biosynthesis Biological Transport Blotting, Western Cell Survival Cells, Cultured Dinoprost - metabolism Enzyme-Linked Immunosorbent Assay Glucose - metabolism Glucose - pharmacokinetics Glucose transport Glucose Transporter Type 1 GLUT1 Hypertrophy Immunohistochemistry Male Mice Mice, Inbred C57BL Monosaccharide Transport Proteins - biosynthesis Monosaccharide Transport Proteins - metabolism Muscle Cells - cytology Myocytes, Cardiac - cytology Oligonucleotides, Antisense - pharmacology Pressure Rats Rats, Sprague-Dawley Subcellular Fractions Survival Time Factors Up-Regulation
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