Endovaginal doppler flow measurements of the ovarian artery in patients with a normal menstrual cycle and with polycystic ovary syndrome during in vitro fertilization
Women with a normal menstrual cycle (n = 21, controls), polycystic ovary syndrome (n = 10) and hypogonadotropic amenorrhea (n = 3) were stimulated with clomiphen‐citrate (4th day to 8th day of the cycle) and with human menopausal gonadotropin (8th day to 11th day). The vascular impedance of the ovar...
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| Veröffentlicht in: | Journal of clinical ultrasound 1993-01, Vol.21 (1), p.19-24 |
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| creator | Schurz, Brigitte Schön, Hans J. Wenzl, Rene Eppel, Wolfgang Huber, Johannes Reinold, Emil |
| description | Women with a normal menstrual cycle (n = 21, controls), polycystic ovary syndrome (n = 10) and hypogonadotropic amenorrhea (n = 3) were stimulated with clomiphen‐citrate (4th day to 8th day of the cycle) and with human menopausal gonadotropin (8th day to 11th day). The vascular impedance of the ovary carrying the dominant follicles was monitored by endovaginal pulsed Doppler flow measurement. Simultaneously, serum levels of LH, E2 and 17‐OHP were assayed. Contrary to controls, women with polycystic ovary syndrome or hypogonadotropic amenorrhea showed decreased hormone levels and no lowering of the vascular impedance. In controls, the lower pulsatility index is caused by neovascularization around the dominant follicle and by E2‐induced vasodilatation in the ovarian artery. © 1993 by John Wiley & Sons. |
| doi_str_mv | 10.1002/jcu.1870210105 |
| format | Article |
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The vascular impedance of the ovary carrying the dominant follicles was monitored by endovaginal pulsed Doppler flow measurement. Simultaneously, serum levels of LH, E2 and 17‐OHP were assayed. Contrary to controls, women with polycystic ovary syndrome or hypogonadotropic amenorrhea showed decreased hormone levels and no lowering of the vascular impedance. In controls, the lower pulsatility index is caused by neovascularization around the dominant follicle and by E2‐induced vasodilatation in the ovarian artery. © 1993 by John Wiley & Sons.</description><identifier>ISSN: 0091-2751</identifier><identifier>EISSN: 1097-0096</identifier><identifier>DOI: 10.1002/jcu.1870210105</identifier><identifier>PMID: 8478440</identifier><identifier>CODEN: JCULDD</identifier><language>eng</language><publisher>New York: Wiley Subscription Services, Inc., A Wiley Company</publisher><subject>17-alpha-Hydroxyprogesterone ; Amenorrhea - diagnostic imaging ; Amenorrhea - physiopathology ; Arteries - diagnostic imaging ; Arteries - physiology ; Biological and medical sciences ; Birth control ; Blood Flow Velocity ; Doppler flow measurement ; Endovaginal ultrasound ; Estradiol - blood ; Female ; Fertilization in Vitro ; Gynecology. Andrology. Obstetrics ; Humans ; Hydroxyprogesterones - blood ; In vitro fertilization ; Luteinizing Hormone - blood ; Medical sciences ; Menstrual Cycle - physiology ; Ovarian artery ; Ovary - blood supply ; Ovary - diagnostic imaging ; Ovary - physiology ; Polycystic ovary syndrome ; Polycystic Ovary Syndrome - diagnostic imaging ; Polycystic Ovary Syndrome - physiopathology ; Sterility. Assisted procreation ; Ultrasonography</subject><ispartof>Journal of clinical ultrasound, 1993-01, Vol.21 (1), p.19-24</ispartof><rights>Copyright © 1993 Wiley Periodicals, Inc., A Wiley Company</rights><rights>1993 INIST-CNRS</rights><lds50>peer_reviewed</lds50><woscitedreferencessubscribed>false</woscitedreferencessubscribed><citedby>FETCH-LOGICAL-c3225-6bdb4f0f54153aba287deda7bd0984fb73dd116d131e748b42df54f53ed08c8c3</citedby><cites>FETCH-LOGICAL-c3225-6bdb4f0f54153aba287deda7bd0984fb73dd116d131e748b42df54f53ed08c8c3</cites></display><links><openurl>$$Topenurl_article</openurl><openurlfulltext>$$Topenurlfull_article</openurlfulltext><thumbnail>$$Tsyndetics_thumb_exl</thumbnail><linktopdf>$$Uhttps://onlinelibrary.wiley.com/doi/pdf/10.1002%2Fjcu.1870210105$$EPDF$$P50$$Gwiley$$H</linktopdf><linktohtml>$$Uhttps://onlinelibrary.wiley.com/doi/full/10.1002%2Fjcu.1870210105$$EHTML$$P50$$Gwiley$$H</linktohtml><link.rule.ids>314,780,784,1416,4022,27922,27923,27924,45573,45574</link.rule.ids><backlink>$$Uhttp://pascal-francis.inist.fr/vibad/index.php?action=getRecordDetail&idt=4524745$$DView record in Pascal Francis$$Hfree_for_read</backlink><backlink>$$Uhttps://www.ncbi.nlm.nih.gov/pubmed/8478440$$D View this record in MEDLINE/PubMed$$Hfree_for_read</backlink></links><search><creatorcontrib>Schurz, Brigitte</creatorcontrib><creatorcontrib>Schön, Hans J.</creatorcontrib><creatorcontrib>Wenzl, Rene</creatorcontrib><creatorcontrib>Eppel, Wolfgang</creatorcontrib><creatorcontrib>Huber, Johannes</creatorcontrib><creatorcontrib>Reinold, Emil</creatorcontrib><title>Endovaginal doppler flow measurements of the ovarian artery in patients with a normal menstrual cycle and with polycystic ovary syndrome during in vitro fertilization</title><title>Journal of clinical ultrasound</title><addtitle>J. Clin. Ultrasound</addtitle><description>Women with a normal menstrual cycle (n = 21, controls), polycystic ovary syndrome (n = 10) and hypogonadotropic amenorrhea (n = 3) were stimulated with clomiphen‐citrate (4th day to 8th day of the cycle) and with human menopausal gonadotropin (8th day to 11th day). The vascular impedance of the ovary carrying the dominant follicles was monitored by endovaginal pulsed Doppler flow measurement. Simultaneously, serum levels of LH, E2 and 17‐OHP were assayed. Contrary to controls, women with polycystic ovary syndrome or hypogonadotropic amenorrhea showed decreased hormone levels and no lowering of the vascular impedance. In controls, the lower pulsatility index is caused by neovascularization around the dominant follicle and by E2‐induced vasodilatation in the ovarian artery. © 1993 by John Wiley & Sons.</description><subject>17-alpha-Hydroxyprogesterone</subject><subject>Amenorrhea - diagnostic imaging</subject><subject>Amenorrhea - physiopathology</subject><subject>Arteries - diagnostic imaging</subject><subject>Arteries - physiology</subject><subject>Biological and medical sciences</subject><subject>Birth control</subject><subject>Blood Flow Velocity</subject><subject>Doppler flow measurement</subject><subject>Endovaginal ultrasound</subject><subject>Estradiol - blood</subject><subject>Female</subject><subject>Fertilization in Vitro</subject><subject>Gynecology. Andrology. Obstetrics</subject><subject>Humans</subject><subject>Hydroxyprogesterones - blood</subject><subject>In vitro fertilization</subject><subject>Luteinizing Hormone - blood</subject><subject>Medical sciences</subject><subject>Menstrual Cycle - physiology</subject><subject>Ovarian artery</subject><subject>Ovary - blood supply</subject><subject>Ovary - diagnostic imaging</subject><subject>Ovary - physiology</subject><subject>Polycystic ovary syndrome</subject><subject>Polycystic Ovary Syndrome - diagnostic imaging</subject><subject>Polycystic Ovary Syndrome - physiopathology</subject><subject>Sterility. Assisted procreation</subject><subject>Ultrasonography</subject><issn>0091-2751</issn><issn>1097-0096</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>1993</creationdate><recordtype>article</recordtype><sourceid>EIF</sourceid><recordid>eNqFkc1u1DAUhSMEKkNhyw7JC8Qug53YcbJEQyk_I9hQys5y_NO6OHZqOx3CA_GceJrRIFasriV_57u2TlE8R3CNIKxe34hpjVoKKwQRJA-KFYIdLSHsmofFKg9UVpSgx8WTGG8ghA0h5KQ4aTFtMYar4veZk_6OXxnHLZB-HK0KQFu_A4PicQpqUC5F4DVI1wpkMhjuAA9JhRkYB0aezD2xM-kacOB8GLIpp2IKUz6JWVgFuJMLMXo7izkmI-5lM4izk8EPCsgpGHe1d96ZFDzQKiRjza-8wLunxSPNbVTPDvO0uHh39nXzvtx-Of-webMtRV1VpGx62WMNNcGI1LznVUulkpz2EnYt1j2tpUSokahGiuK2x5XMrCa1krAVrahPi1eLdwz-dlIxscFEoazlTvkpMkqaDpOWZHC9gCL4GIPSbAxmyB9iCLJ9MSwXw_4WkwMvDuapH5Q84ocm8v3Lwz2PglsduBMmHjFMKkzxXtMt2M5YNf9nKfu4ufjnCeWSNTGpn8csDz9YQ2tK2OXnc0Yu4bf609vvbFv_AdTtuz4</recordid><startdate>199301</startdate><enddate>199301</enddate><creator>Schurz, Brigitte</creator><creator>Schön, Hans J.</creator><creator>Wenzl, Rene</creator><creator>Eppel, Wolfgang</creator><creator>Huber, Johannes</creator><creator>Reinold, Emil</creator><general>Wiley Subscription Services, Inc., A Wiley Company</general><general>Wiley</general><scope>BSCLL</scope><scope>IQODW</scope><scope>CGR</scope><scope>CUY</scope><scope>CVF</scope><scope>ECM</scope><scope>EIF</scope><scope>NPM</scope><scope>AAYXX</scope><scope>CITATION</scope><scope>7X8</scope></search><sort><creationdate>199301</creationdate><title>Endovaginal doppler flow measurements of the ovarian artery in patients with a normal menstrual cycle and with polycystic ovary syndrome during in vitro fertilization</title><author>Schurz, Brigitte ; Schön, Hans J. ; Wenzl, Rene ; Eppel, Wolfgang ; Huber, Johannes ; Reinold, Emil</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-c3225-6bdb4f0f54153aba287deda7bd0984fb73dd116d131e748b42df54f53ed08c8c3</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng</language><creationdate>1993</creationdate><topic>17-alpha-Hydroxyprogesterone</topic><topic>Amenorrhea - diagnostic imaging</topic><topic>Amenorrhea - physiopathology</topic><topic>Arteries - diagnostic imaging</topic><topic>Arteries - physiology</topic><topic>Biological and medical sciences</topic><topic>Birth control</topic><topic>Blood Flow Velocity</topic><topic>Doppler flow measurement</topic><topic>Endovaginal ultrasound</topic><topic>Estradiol - blood</topic><topic>Female</topic><topic>Fertilization in Vitro</topic><topic>Gynecology. Andrology. Obstetrics</topic><topic>Humans</topic><topic>Hydroxyprogesterones - blood</topic><topic>In vitro fertilization</topic><topic>Luteinizing Hormone - blood</topic><topic>Medical sciences</topic><topic>Menstrual Cycle - physiology</topic><topic>Ovarian artery</topic><topic>Ovary - blood supply</topic><topic>Ovary - diagnostic imaging</topic><topic>Ovary - physiology</topic><topic>Polycystic ovary syndrome</topic><topic>Polycystic Ovary Syndrome - diagnostic imaging</topic><topic>Polycystic Ovary Syndrome - physiopathology</topic><topic>Sterility. Assisted procreation</topic><topic>Ultrasonography</topic><toplevel>peer_reviewed</toplevel><toplevel>online_resources</toplevel><creatorcontrib>Schurz, Brigitte</creatorcontrib><creatorcontrib>Schön, Hans J.</creatorcontrib><creatorcontrib>Wenzl, Rene</creatorcontrib><creatorcontrib>Eppel, Wolfgang</creatorcontrib><creatorcontrib>Huber, Johannes</creatorcontrib><creatorcontrib>Reinold, Emil</creatorcontrib><collection>Istex</collection><collection>Pascal-Francis</collection><collection>Medline</collection><collection>MEDLINE</collection><collection>MEDLINE (Ovid)</collection><collection>MEDLINE</collection><collection>MEDLINE</collection><collection>PubMed</collection><collection>CrossRef</collection><collection>MEDLINE - Academic</collection><jtitle>Journal of clinical ultrasound</jtitle></facets><delivery><delcategory>Remote Search Resource</delcategory><fulltext>fulltext</fulltext></delivery><addata><au>Schurz, Brigitte</au><au>Schön, Hans J.</au><au>Wenzl, Rene</au><au>Eppel, Wolfgang</au><au>Huber, Johannes</au><au>Reinold, Emil</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>Endovaginal doppler flow measurements of the ovarian artery in patients with a normal menstrual cycle and with polycystic ovary syndrome during in vitro fertilization</atitle><jtitle>Journal of clinical ultrasound</jtitle><addtitle>J. Clin. Ultrasound</addtitle><date>1993-01</date><risdate>1993</risdate><volume>21</volume><issue>1</issue><spage>19</spage><epage>24</epage><pages>19-24</pages><issn>0091-2751</issn><eissn>1097-0096</eissn><coden>JCULDD</coden><abstract>Women with a normal menstrual cycle (n = 21, controls), polycystic ovary syndrome (n = 10) and hypogonadotropic amenorrhea (n = 3) were stimulated with clomiphen‐citrate (4th day to 8th day of the cycle) and with human menopausal gonadotropin (8th day to 11th day). The vascular impedance of the ovary carrying the dominant follicles was monitored by endovaginal pulsed Doppler flow measurement. Simultaneously, serum levels of LH, E2 and 17‐OHP were assayed. Contrary to controls, women with polycystic ovary syndrome or hypogonadotropic amenorrhea showed decreased hormone levels and no lowering of the vascular impedance. In controls, the lower pulsatility index is caused by neovascularization around the dominant follicle and by E2‐induced vasodilatation in the ovarian artery. © 1993 by John Wiley & Sons.</abstract><cop>New York</cop><pub>Wiley Subscription Services, Inc., A Wiley Company</pub><pmid>8478440</pmid><doi>10.1002/jcu.1870210105</doi><tpages>6</tpages></addata></record> |
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| subjects | 17-alpha-Hydroxyprogesterone Amenorrhea - diagnostic imaging Amenorrhea - physiopathology Arteries - diagnostic imaging Arteries - physiology Biological and medical sciences Birth control Blood Flow Velocity Doppler flow measurement Endovaginal ultrasound Estradiol - blood Female Fertilization in Vitro Gynecology. Andrology. Obstetrics Humans Hydroxyprogesterones - blood In vitro fertilization Luteinizing Hormone - blood Medical sciences Menstrual Cycle - physiology Ovarian artery Ovary - blood supply Ovary - diagnostic imaging Ovary - physiology Polycystic ovary syndrome Polycystic Ovary Syndrome - diagnostic imaging Polycystic Ovary Syndrome - physiopathology Sterility. Assisted procreation Ultrasonography |
| title | Endovaginal doppler flow measurements of the ovarian artery in patients with a normal menstrual cycle and with polycystic ovary syndrome during in vitro fertilization |
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