Uptake of apoptotic cells drives the growth of a pathogenic trypanosome in macrophages

After apoptosis, phagocytes prevent inflammation and tissue damage by the uptake and removal of dead cells 1 . In addition, apoptotic cells evoke an anti-inflammatory response through macrophages 2 , 3 . We have previously shown that there is intense lymphocyte apoptosis in an experimental model of...

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Veröffentlicht in:	Nature (London) 2000-01, Vol.403 (6766), p.199-203
Hauptverfasser:	Freire-de-Lima, Célio G., Nascimento, Danielle O., Soares, Milena B. P., Bozza, Patricia T., Castro-Faria-Neto, Hugo C., de Mello, Fernando G., DosReis, George A., Lopes, Marcela F.
Format:	Artikel
Sprache:	eng
Schlagworte:	Amino Acid Chloromethyl Ketones - pharmacology Animals Apoptosis Bacteria Biosynthesis Cells Cells, Cultured Chagas Disease - immunology Chagas Disease - parasitology cyclooxygenase inhibitors Cysteine Proteinase Inhibitors - pharmacology Dinoprostone - biosynthesis Dinoprostone - physiology Disease Humanities and Social Sciences letter Lymphocytes Macrophages - parasitology Male Mice Mice, Inbred BALB C multidisciplinary Necrosis Parasite control Phagocytosis - physiology Putrescine - biosynthesis Putrescine - physiology Receptors, Vitronectin - metabolism Rodents Science Science (multidisciplinary) T-Lymphocytes - drug effects T-Lymphocytes - pathology T-Lymphocytes - physiology Transforming Growth Factor beta - physiology Trypanosoma cruzi Trypanosoma cruzi - growth & development Vector-borne diseases vitronectin receptors
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