Tamoxifen, protein kinase C and rat sperm mitochondria

Tamoxifen at a dose of 400 μg/kg/day has been reported to reduce the fertility of adult male rats and alter the pattern of cauda sperm motility from forward progressive to circular yawing type. Since sperm motility is powered by mitochondria, the effect of tamoxifen on mitochondrial function was stu...

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Veröffentlicht in:	Cell biology international 2003-09, Vol.27 (9), p.761-768
Hauptverfasser:	Saberwal, Gurveen Sethi, Gill-Sharma, Manjit K., Balasinor, Nafisa, Choudhary, Jyoti, Padwal, Varsha, Juneja, H.S.
Format:	Artikel
Sprache:	eng
Schlagworte:	Adenosine Triphosphate - metabolism Animals Biological Transport Creatine Kinase - metabolism Fluorescent Dyes - metabolism Glycolysis Male Malondialdehyde - metabolism Mitochondria Mitochondria - drug effects Mitochondria - metabolism Mitochondria - ultrastructure Protein Kinase C Protein Kinase C - metabolism Rats Rhodamine 123 Rhodamine 123 - metabolism Selective Estrogen Receptor Modulators - pharmacology Sperm motility Sperm Motility - drug effects Spermatozoa - drug effects Spermatozoa - enzymology Spermatozoa - ultrastructure Tamoxifen Tamoxifen - pharmacology
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