Parasympathetic failure and risk of subsequent coronary events in unstable angina and non-ST-segment elevation myocardial infarction

Parasympathetic failure and risk of subsequent coronary events in unstable angina and non-ST-segment elevation myocardial infarction

Aim Previous animal studies suggested that vagal tone contributes to tonic dilatation of coronary arteries. We hypothesized that low parasympathetic activity might be among the causes of coronary instability in the setting of acute coronary syndrome without ST-segment elevation. Methods and results...

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Veröffentlicht in:European heart journal 2003-09, Vol.24 (17), p.1560-1566
Hauptverfasser: Manfrini, Olivia, Pizzi, Carmine, Trerè, Davide, Fontana, Fiorella, Bugiardini, Raffaele
Format: Artikel
Sprache:eng
Schlagworte:
Angina, Unstable - etiology
Arrhythmias, Cardiac - etiology
Arrhythmias, Cardiac - therapy
Autonomic Nervous System Diseases - complications
Biological and medical sciences
Cardiology. Vascular system
Coronary Care Units
Coronary heart disease
Disease-Free Survival
Female
Heart
Heart rate variability
Hospitalization
Humans
Male
Medical sciences
Middle Aged
Multivariate Analysis
Myocardial Infarction - etiology
Myocardial Ischemia - etiology
Myocarditis. Cardiomyopathies
Parasympathetic Nervous System
Prognosis
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Zusammenfassung:Aim Previous animal studies suggested that vagal tone contributes to tonic dilatation of coronary arteries. We hypothesized that low parasympathetic activity might be among the causes of coronary instability in the setting of acute coronary syndrome without ST-segment elevation. Methods and results We studied 172 consecutive patients. Vagal and sympathetic activities were assessed by time domain measures of heart rate variability. PNN50
ISSN:0195-668X
1522-9645
DOI:10.1016/S0195-668X(03)00345-2