Q-T Peak Dispersion in Congenital Long QT Syndrome: Possible Marker of Mutation of HERG

Congenital long QT syndrome (LQTS) is caused by mutations in various cardiac potassium or sodium channel genes, with 6 different genotypes thus far identified. However, it is unknown whether these genotypes can be differentiated by QT variables. The electrocardiograms obtained from 16 patients with...

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Veröffentlicht in:	Circulation Journal 2003, Vol.67(6), pp.495-498
Hauptverfasser:	Inoue, Masaru, Shimizu, Masami, Ino, Hidekazu, Yamaguchi, Masato, Terai, Hidenobu, Hayashi, Kenshi, Kiyama, Masaru, Sakata, Kenji, Hayashi, Tatsumi, Mabuchi, Hiroshi
Format:	Artikel
Sprache:	eng
Schlagworte:	Adult Amino Acid Substitution Biological and medical sciences Cardiac dysrhythmias Cardiology. Vascular system Cation Transport Proteins DNA-Binding Proteins Electrocardiography ERG1 Potassium Channel Ether-A-Go-Go Potassium Channels Female Heart Humans KCNQ Potassium Channels KCNQ1 Potassium Channel Long QT syndrome Long QT Syndrome - classification Long QT Syndrome - congenital Long QT Syndrome - genetics Long QT Syndrome - physiopathology Male Medical sciences Middle Aged Mutation, Missense Notched T wave Potassium Channels - deficiency Potassium Channels - genetics Potassium Channels - physiology Potassium Channels, Voltage-Gated QT dispersion Trans-Activators Transcriptional Regulator ERG
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