N-cadherin is depleted from proximal tubules in experimental and human acute kidney injury

Ischemia remains the most common cause of acute kidney injury (AKI). Decreased intercellular adhesion and alterations in adhesion molecules may contribute to the loss of renal function observed in AKI. In the present study, we evaluated the distribution of adhesion molecules in the human kidney and...

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Veröffentlicht in:	Histochemistry and cell biology 2010-06, Vol.133 (6), p.641-649
Hauptverfasser:	Nürnberger, Jens, Feldkamp, Thorsten, Kavapurackal, Rosmaria, Opazo Saez, Anabelle, Becker, Jan, Hörbelt, Markus, Kribben, Andreas
Format:	Artikel
Sprache:	eng
Schlagworte:	Acute kidney injury Acute Kidney Injury - physiopathology Adhesion junction Animals Biochemistry Biomedical and Life Sciences Biomedicine Cadherin Cadherins - chemistry Cadherins - classification Cadherins - metabolism Cell adhesion & migration Cell Biology Cells, Cultured Developmental Biology Disease Models, Animal Humans Immunohistochemistry Injuries Ischemia Kidney - injuries Kidney - metabolism Kidney - pathology Kidney Tubules, Proximal - metabolism Kidneys Male Membranes Opossums Original Paper Proteins Rats Rats, Sprague-Dawley
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