Successful Treatment of Severe Orthostatic Hypotension with Erythropoietin
KAWAKAMI, K., et al.: Successful Treatment of Severe Orthostatic Hypotension with Erythropoietin. A 71‐year‐old man, who was diagnosed with familial amyloidosis type I, was admitted for treatment of severe orthostatic hypotension associated with recurrent syncopal attacks. Head‐up tilt testing demon...
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| Veröffentlicht in: | Pacing and clinical electrophysiology 2003-01, Vol.26 (1p1), p.105-107 |
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| container_issue | 1p1 |
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| container_title | Pacing and clinical electrophysiology |
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| creator | KAWAKAMI, KAZUNOBU ABE, HARUHIKO HARAYAMA, NOBUYA NAKASHIMA, YASUHIDE |
| description | KAWAKAMI, K., et al.: Successful Treatment of Severe Orthostatic Hypotension with Erythropoietin.
A 71‐year‐old man, who was diagnosed with familial amyloidosis type I, was admitted for treatment of severe orthostatic hypotension associated with recurrent syncopal attacks. Head‐up tilt testing demonstrated severe orthostatic hypotension (114/72 mmHg in the supine position and 62/34 mmHg in the upright position) with syncope or presyncope. Oral midodorine and fludrocortisone therapies failed to prevent his symptoms. After administration of subcutaneous erythropoietin, his blood pressure drop in the upright position was decreased and symptoms disappeared unassociated with improvement of anemia. Although previous reports have shown that the mechanism by which erythropoietin improves orthostatic hypotension is related to improvement in anemia, other mechanisms may also play a role. (PACE 2003; 26[Pt. I]:105–107) |
| doi_str_mv | 10.1046/j.1460-9592.2003.00158.x |
| format | Article |
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A 71‐year‐old man, who was diagnosed with familial amyloidosis type I, was admitted for treatment of severe orthostatic hypotension associated with recurrent syncopal attacks. Head‐up tilt testing demonstrated severe orthostatic hypotension (114/72 mmHg in the supine position and 62/34 mmHg in the upright position) with syncope or presyncope. Oral midodorine and fludrocortisone therapies failed to prevent his symptoms. After administration of subcutaneous erythropoietin, his blood pressure drop in the upright position was decreased and symptoms disappeared unassociated with improvement of anemia. Although previous reports have shown that the mechanism by which erythropoietin improves orthostatic hypotension is related to improvement in anemia, other mechanisms may also play a role. (PACE 2003; 26[Pt. I]:105–107)</description><identifier>ISSN: 0147-8389</identifier><identifier>EISSN: 1540-8159</identifier><identifier>DOI: 10.1046/j.1460-9592.2003.00158.x</identifier><identifier>PMID: 12685148</identifier><language>eng</language><publisher>350 Main Street , Malden , MA 02148-5018 , U.S.A: Blackwell Futura Publishing, Inc</publisher><subject>Aged ; Amyloid Neuropathies, Familial - complications ; Blood Pressure - drug effects ; erythropoietin ; Erythropoietin - therapeutic use ; head-up tilt test ; Humans ; Hypotension, Orthostatic - drug therapy ; Hypotension, Orthostatic - etiology ; Male ; orthostatic hypotension ; Recombinant Proteins ; syncope ; Syncope - etiology ; Tilt-Table Test</subject><ispartof>Pacing and clinical electrophysiology, 2003-01, Vol.26 (1p1), p.105-107</ispartof><lds50>peer_reviewed</lds50><woscitedreferencessubscribed>false</woscitedreferencessubscribed><citedby>FETCH-LOGICAL-c4038-538bfb296e7836894639d8f233a6a8e877358261c2e626491977edfddeda44993</citedby><cites>FETCH-LOGICAL-c4038-538bfb296e7836894639d8f233a6a8e877358261c2e626491977edfddeda44993</cites></display><links><openurl>$$Topenurl_article</openurl><openurlfulltext>$$Topenurlfull_article</openurlfulltext><thumbnail>$$Tsyndetics_thumb_exl</thumbnail><linktopdf>$$Uhttps://onlinelibrary.wiley.com/doi/pdf/10.1046%2Fj.1460-9592.2003.00158.x$$EPDF$$P50$$Gwiley$$H</linktopdf><linktohtml>$$Uhttps://onlinelibrary.wiley.com/doi/full/10.1046%2Fj.1460-9592.2003.00158.x$$EHTML$$P50$$Gwiley$$H</linktohtml><link.rule.ids>314,780,784,1417,27924,27925,45574,45575</link.rule.ids><backlink>$$Uhttps://www.ncbi.nlm.nih.gov/pubmed/12685148$$D View this record in MEDLINE/PubMed$$Hfree_for_read</backlink></links><search><creatorcontrib>KAWAKAMI, KAZUNOBU</creatorcontrib><creatorcontrib>ABE, HARUHIKO</creatorcontrib><creatorcontrib>HARAYAMA, NOBUYA</creatorcontrib><creatorcontrib>NAKASHIMA, YASUHIDE</creatorcontrib><title>Successful Treatment of Severe Orthostatic Hypotension with Erythropoietin</title><title>Pacing and clinical electrophysiology</title><addtitle>Pacing Clin Electrophysiol</addtitle><description>KAWAKAMI, K., et al.: Successful Treatment of Severe Orthostatic Hypotension with Erythropoietin.
A 71‐year‐old man, who was diagnosed with familial amyloidosis type I, was admitted for treatment of severe orthostatic hypotension associated with recurrent syncopal attacks. Head‐up tilt testing demonstrated severe orthostatic hypotension (114/72 mmHg in the supine position and 62/34 mmHg in the upright position) with syncope or presyncope. Oral midodorine and fludrocortisone therapies failed to prevent his symptoms. After administration of subcutaneous erythropoietin, his blood pressure drop in the upright position was decreased and symptoms disappeared unassociated with improvement of anemia. Although previous reports have shown that the mechanism by which erythropoietin improves orthostatic hypotension is related to improvement in anemia, other mechanisms may also play a role. (PACE 2003; 26[Pt. I]:105–107)</description><subject>Aged</subject><subject>Amyloid Neuropathies, Familial - complications</subject><subject>Blood Pressure - drug effects</subject><subject>erythropoietin</subject><subject>Erythropoietin - therapeutic use</subject><subject>head-up tilt test</subject><subject>Humans</subject><subject>Hypotension, Orthostatic - drug therapy</subject><subject>Hypotension, Orthostatic - etiology</subject><subject>Male</subject><subject>orthostatic hypotension</subject><subject>Recombinant Proteins</subject><subject>syncope</subject><subject>Syncope - etiology</subject><subject>Tilt-Table Test</subject><issn>0147-8389</issn><issn>1540-8159</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>2003</creationdate><recordtype>article</recordtype><sourceid>EIF</sourceid><recordid>eNqNkE1P20AQQFcVqKS0f6HyqTe7-_1x6AGiAC0IWkHV3lYbe6xscLxhd12Sf1-HRPTa08zhvRnpIVQQXBHM5edlRbjEpRGGVhRjVmFMhK42b9CECI5LTYQ5QhNMuCo10-YEvUtpiTGWmIu36IRQqQXheoK-3Q91DSm1Q1c8RHB5BX0uQlvcwx-IUNzFvAgpu-zr4mq7Dhn65ENfPPu8KGZxmxcxrIOH7Pv36Lh1XYIPh3mKfl7MHqZX5c3d5dfp2U1Zc8x0KZiet3NqJCjNpDZcMtPoljLmpNOglWJCU0lqCpJKbohRCpq2aaBxnBvDTtGn_d11DE8DpGxXPtXQda6HMCSrGB0bCTWCeg_WMaQUobXr6Fcubi3BdtfRLu2uo911tLuO9qWj3Yzqx8OPYb6C5p94CDcCX_bAs-9g-9-H7fez6WzcRr_c-z5l2Lz6Lj5aqZgS9tftpZ1enJNr_lvbH-wvJ7CQbA</recordid><startdate>200301</startdate><enddate>200301</enddate><creator>KAWAKAMI, KAZUNOBU</creator><creator>ABE, HARUHIKO</creator><creator>HARAYAMA, NOBUYA</creator><creator>NAKASHIMA, YASUHIDE</creator><general>Blackwell Futura Publishing, Inc</general><scope>BSCLL</scope><scope>CGR</scope><scope>CUY</scope><scope>CVF</scope><scope>ECM</scope><scope>EIF</scope><scope>NPM</scope><scope>AAYXX</scope><scope>CITATION</scope><scope>7X8</scope></search><sort><creationdate>200301</creationdate><title>Successful Treatment of Severe Orthostatic Hypotension with Erythropoietin</title><author>KAWAKAMI, KAZUNOBU ; ABE, HARUHIKO ; HARAYAMA, NOBUYA ; NAKASHIMA, YASUHIDE</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-c4038-538bfb296e7836894639d8f233a6a8e877358261c2e626491977edfddeda44993</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng</language><creationdate>2003</creationdate><topic>Aged</topic><topic>Amyloid Neuropathies, Familial - complications</topic><topic>Blood Pressure - drug effects</topic><topic>erythropoietin</topic><topic>Erythropoietin - therapeutic use</topic><topic>head-up tilt test</topic><topic>Humans</topic><topic>Hypotension, Orthostatic - drug therapy</topic><topic>Hypotension, Orthostatic - etiology</topic><topic>Male</topic><topic>orthostatic hypotension</topic><topic>Recombinant Proteins</topic><topic>syncope</topic><topic>Syncope - etiology</topic><topic>Tilt-Table Test</topic><toplevel>peer_reviewed</toplevel><toplevel>online_resources</toplevel><creatorcontrib>KAWAKAMI, KAZUNOBU</creatorcontrib><creatorcontrib>ABE, HARUHIKO</creatorcontrib><creatorcontrib>HARAYAMA, NOBUYA</creatorcontrib><creatorcontrib>NAKASHIMA, YASUHIDE</creatorcontrib><collection>Istex</collection><collection>Medline</collection><collection>MEDLINE</collection><collection>MEDLINE (Ovid)</collection><collection>MEDLINE</collection><collection>MEDLINE</collection><collection>PubMed</collection><collection>CrossRef</collection><collection>MEDLINE - Academic</collection><jtitle>Pacing and clinical electrophysiology</jtitle></facets><delivery><delcategory>Remote Search Resource</delcategory><fulltext>fulltext</fulltext></delivery><addata><au>KAWAKAMI, KAZUNOBU</au><au>ABE, HARUHIKO</au><au>HARAYAMA, NOBUYA</au><au>NAKASHIMA, YASUHIDE</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>Successful Treatment of Severe Orthostatic Hypotension with Erythropoietin</atitle><jtitle>Pacing and clinical electrophysiology</jtitle><addtitle>Pacing Clin Electrophysiol</addtitle><date>2003-01</date><risdate>2003</risdate><volume>26</volume><issue>1p1</issue><spage>105</spage><epage>107</epage><pages>105-107</pages><issn>0147-8389</issn><eissn>1540-8159</eissn><abstract>KAWAKAMI, K., et al.: Successful Treatment of Severe Orthostatic Hypotension with Erythropoietin.
A 71‐year‐old man, who was diagnosed with familial amyloidosis type I, was admitted for treatment of severe orthostatic hypotension associated with recurrent syncopal attacks. Head‐up tilt testing demonstrated severe orthostatic hypotension (114/72 mmHg in the supine position and 62/34 mmHg in the upright position) with syncope or presyncope. Oral midodorine and fludrocortisone therapies failed to prevent his symptoms. After administration of subcutaneous erythropoietin, his blood pressure drop in the upright position was decreased and symptoms disappeared unassociated with improvement of anemia. Although previous reports have shown that the mechanism by which erythropoietin improves orthostatic hypotension is related to improvement in anemia, other mechanisms may also play a role. (PACE 2003; 26[Pt. I]:105–107)</abstract><cop>350 Main Street , Malden , MA 02148-5018 , U.S.A</cop><pub>Blackwell Futura Publishing, Inc</pub><pmid>12685148</pmid><doi>10.1046/j.1460-9592.2003.00158.x</doi><tpages>3</tpages></addata></record> |
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| source | MEDLINE; Access via Wiley Online Library |
| subjects | Aged Amyloid Neuropathies, Familial - complications Blood Pressure - drug effects erythropoietin Erythropoietin - therapeutic use head-up tilt test Humans Hypotension, Orthostatic - drug therapy Hypotension, Orthostatic - etiology Male orthostatic hypotension Recombinant Proteins syncope Syncope - etiology Tilt-Table Test |
| title | Successful Treatment of Severe Orthostatic Hypotension with Erythropoietin |
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