Hemorrhage-induced vascular hyporeactivity to norepinephrine in select vasculatures of rats and the roles of nitric oxide and endothelin

Hemorrhage-induced vascular hyporeactivity to norepinephrine (NE) and the possible effector roles of nitric oxide (NO) and endothelin (ET) were investigated in different vascular beds of rats. Under urethane anesthesia, rats (n = 7 per group) were hemorrhaged to a mean arterial pressure (MAP) of 50...

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Veröffentlicht in:	Shock (Augusta, Ga.) Ga.), 2003-03, Vol.19 (3), p.208-214
Hauptverfasser:	LIU, Liang-Ming, WARD, John A, DUBICK, Michael A
Format:	Artikel
Sprache:	eng
Schlagworte:	Anesthesia. Intensive care medicine. Transfusions. Cell therapy and gene therapy Animals Biological and medical sciences Blood Flow Velocity - drug effects Blood Flow Velocity - physiology Blood Pressure Emergency and intensive cardiocirculatory care. Cardiogenic shock. Coronary intensive care Endothelin Receptor Antagonists Endothelins - drug effects Endothelins - physiology Hemorrhage - physiopathology Intensive care medicine Male Medical sciences Muscle, Smooth, Vascular - drug effects Muscle, Smooth, Vascular - physiopathology NG-Nitroarginine Methyl Ester - pharmacology Nitric Oxide - physiology Nitric Oxide Synthase - antagonists & inhibitors Norepinephrine - pharmacology Oligopeptides - pharmacology Rats Rats, Sprague-Dawley Reference Values Time Factors
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