The antidepressants fluoxetine, idazoxan and phenelzine alter corticotropin-releasing hormone and tyrosine hydroxylase mRNA levels in rat brain: therapeutic implications

Various classes of antidepressant drugs with distinct pharmacologic actions are differentially effective in the treatment of classic melancholic depression — characterized by pathological hyperarousal and atypical depression — associated with lethargy, hypersomnia, and hyperphagia. All antidepressan...

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Veröffentlicht in:	Brain research 1992-02, Vol.572 (1), p.117-125
Hauptverfasser:	Brady, Linda S., Gold, Philip W., Herkenham, Miles, Lynn, Allison B., Whitfield, Harvey J.
Format:	Artikel
Sprache:	eng
Schlagworte:	Adrenergic alpha-Antagonists - pharmacology Animals Antidepressant drug Antidepressive Agents - pharmacology Biological and medical sciences Corticotropin-releasing hormone Corticotropin-Releasing Hormone - genetics Depression Dioxanes - pharmacology Fluoxetine - pharmacology Glucocorticoid receptor Idazoxan In situ hybridization Locus coeruleus Locus Coeruleus - drug effects Locus Coeruleus - metabolism Male Medical sciences Mineralcorticoid receptor Mineralocorticoids Neuropharmacology Paraventricular Hypothalamic Nucleus - drug effects Paraventricular Hypothalamic Nucleus - metabolism Paraventricular nucleus Pharmacology. Drug treatments Phenelzine - pharmacology Psychoanaleptics: cns stimulant, antidepressant agent, nootropic agent, mood stabilizer..., (alzheimer disease) Psychology. Psychoanalysis. Psychiatry Psychopharmacology Rats Rats, Inbred Strains Receptors, Glucocorticoid - genetics Receptors, Mineralocorticoid Receptors, Steroid - genetics RNA, Messenger - genetics RNA, Messenger - metabolism Tyrosine 3-Monooxygenase - genetics Tyrosine hydroxylase
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