Short Chain Fatty Acids and Colon Cancer

The development of intestinal cancer involves complex genetic and epigenetic alterations in the intestinal mucosa. The principal signaling pathway responsible for the initiation of tumor formation, the APC-β-catenin-TCF4 pathway, regulates both cell proliferation and colonic cell differentiation, bu...

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Veröffentlicht in:	The Journal of nutrition 2002-12, Vol.132 (12), p.3804S-3808S
Hauptverfasser:	Augenlicht, Leonard H., Mariadason, John M., Wilson, Andrew, Arango, Diego, Yang, WanCai, Heerdt, Barbara G., Velcich, Anna
Format:	Artikel
Sprache:	eng
Schlagworte:	adenocarcinoma Animals apoptosis beta Catenin Cancer Cell Differentiation cell maturation cell movement cell proliferation Cells Colonic Neoplasms - genetics Colonic Neoplasms - metabolism colorectal neoplasms Cytoskeletal Proteins - metabolism Disease Models, Animal drugs epigenetics Fatty Acids - metabolism gene expression Gene Expression Profiling genes Genetics goblet cells Humans intestinal cancer intestinal mucosa Mice mouse genetic models mucin mucins Mucins - metabolism Nutrition Oligonucleotide Array Sequence Analysis rectum Rodents short chain fatty acids Trans-Activators - metabolism Transcription Factors - metabolism Transcription, Genetic
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