Analysis of the effects of activation of the alternative pathway of complement on erythrocytes with an isolated deficiency of decay accelerating factor

E from individuals with the Inab blood group phenotype have an isolated deficiency of decay accelerating factor (DAF, CD55). DAF is a glycosyl phosphatidylinositol anchored membrane protein that inhibits activation of both the classical and alternative pathways of complement. Deficiency of DAF from...

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Veröffentlicht in:The Journal of immunology (1950) 1992-01, Vol.148 (2), p.498-502
Hauptverfasser: Holguin, MH, Martin, CB, Bernshaw, NJ, Parker, CJ
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Sprache:eng
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