Acetylcholine receptor regulation in L5 muscle cells is independent of increases in collagen secretion induced by ascorbic acid

Ascorbic acid is the active component of fetal brain extract that induces increased acetylcholine receptor (AChR) expression in L5 rat clonal muscle cell cultures. The induction of AChR expression, as determined by125I-α-bun-garotoxin binding, occurs with a delay of 20–25 h. We report that the delay...

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Veröffentlicht in:	The American journal of clinical nutrition 1991-12, Vol.54 (6), p.1184S-1187S
Hauptverfasser:	Salpeter, MM, Liu, EC, Minor, RR, Podleski, TR, Wootton, JA
Format:	Artikel
Sprache:	eng
Schlagworte:	acetylcholine acetylcholine receptor Animals Antineoplastic agents Ascorbic acid Ascorbic Acid - pharmacology Biological and medical sciences Cell Line cells cells in vitro collagen Collagen - secretion General aspects Medical sciences muscle muscles Muscles - cytology Muscles - metabolism Pharmacology. Drug treatments receptors Receptors, Cholinergic - genetics Receptors, Cholinergic - metabolism RNA, Messenger - metabolism secretion
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