Pulmonary oxygen consumption : a hypothesis to explain the increase in oxygen consumption of low birth weight infants with lung disease
We determined pulmonary oxygen consumption (VO2lung) in low-birthweight infants with acute lung disease to help explain the greater whole-body oxygen consumption (VO2wb) in these infants with than in those without lung disease. Eleven infants (birth weight 1,076+/-364 g; gestational age 28+/-3 weeks...
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| Veröffentlicht in: | Intensive care medicine 2001-10, Vol.27 (10), p.1636-1642 |
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| creator | SCHULZE, Andreas ABUBAKAR, Kabir GILL, Gerald WAY, R. Clifton SINCLAIR, John C |
| description | We determined pulmonary oxygen consumption (VO2lung) in low-birthweight infants with acute lung disease to help explain the greater whole-body oxygen consumption (VO2wb) in these infants with than in those without lung disease.
Eleven infants (birth weight 1,076+/-364 g; gestational age 28+/-3 weeks) undergoing mechanical ventilation for respiratory distress syndrome were studied in their first week of life. We measured VO2wb by indirect calorimetry and simultaneously determined systemic oxygen uptake (VO2Fick) as the product of cardiac output (echocardiography) and the arterial-mixed venous oxygen content difference (cooximetry) assuming that VO2wb-VO2Fick accounts for VO2lung. Right atrial blood samples were used to determine mixed venous oxygenation, and infants were excluded if samples returned saturations greater than 89%.
VO2lung was 1.92+/-1.74 ml x kg(-1) x min(-1), representing 25% of their VO2wb (7.58+/-1.48 ml x kg(-1) x min(-1)). VO2lung was not correlated with clinical measures of acute disease severity. However, infants with the most severe changes on follow-up radiography (Edwards score 5 as assessed by radiologist blinded for VO2 data) all had a VO2lung level greater than 2.0 ml x kg(-1) x min(-1).
VO2lung can account for the elevated metabolic rate in low-birthweight infants with lung injury. We speculate that this reflects in part inflammatory pulmonary processes and may herald chronic lung disease. |
| doi_str_mv | 10.1007/s001340101074 |
| format | Article |
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Eleven infants (birth weight 1,076+/-364 g; gestational age 28+/-3 weeks) undergoing mechanical ventilation for respiratory distress syndrome were studied in their first week of life. We measured VO2wb by indirect calorimetry and simultaneously determined systemic oxygen uptake (VO2Fick) as the product of cardiac output (echocardiography) and the arterial-mixed venous oxygen content difference (cooximetry) assuming that VO2wb-VO2Fick accounts for VO2lung. Right atrial blood samples were used to determine mixed venous oxygenation, and infants were excluded if samples returned saturations greater than 89%.
VO2lung was 1.92+/-1.74 ml x kg(-1) x min(-1), representing 25% of their VO2wb (7.58+/-1.48 ml x kg(-1) x min(-1)). VO2lung was not correlated with clinical measures of acute disease severity. However, infants with the most severe changes on follow-up radiography (Edwards score 5 as assessed by radiologist blinded for VO2 data) all had a VO2lung level greater than 2.0 ml x kg(-1) x min(-1).
VO2lung can account for the elevated metabolic rate in low-birthweight infants with lung injury. We speculate that this reflects in part inflammatory pulmonary processes and may herald chronic lung disease.</description><identifier>ISSN: 0342-4642</identifier><identifier>EISSN: 1432-1238</identifier><identifier>DOI: 10.1007/s001340101074</identifier><identifier>PMID: 11685305</identifier><identifier>CODEN: ICMED9</identifier><language>eng</language><publisher>Heidelberg: Springer</publisher><subject>Acute Disease ; Anesthesia. Intensive care medicine. Transfusions. Cell therapy and gene therapy ; Biological and medical sciences ; Birth Weight ; Calorimetry, Indirect ; Carbon Dioxide - blood ; Cardiac Output ; Chronic Disease ; Emergency and intensive respiratory care ; Energy Metabolism ; Female ; Gestational Age ; Humans ; Hyaline Membrane Disease - diagnostic imaging ; Hyaline Membrane Disease - immunology ; Hyaline Membrane Disease - metabolism ; Hyaline Membrane Disease - physiopathology ; Hyaline Membrane Disease - therapy ; Hypotheses ; Infant, Low Birth Weight ; Infant, Newborn ; Infant, Premature, Diseases - diagnostic imaging ; Infant, Premature, Diseases - immunology ; Infant, Premature, Diseases - metabolism ; Infant, Premature, Diseases - physiopathology ; Infant, Premature, Diseases - therapy ; Inflammation ; Intensive care medicine ; Lung - metabolism ; Lung diseases ; Lung Diseases - diagnostic imaging ; Lung Diseases - immunology ; Lung Diseases - metabolism ; Lung Diseases - physiopathology ; Lung Diseases - therapy ; Male ; Medical sciences ; Oximetry ; Oxygen - blood ; Oxygen Consumption ; Radiography ; Respiration, Artificial ; Severity of Illness Index ; Single-Blind Method ; Time Factors</subject><ispartof>Intensive care medicine, 2001-10, Vol.27 (10), p.1636-1642</ispartof><rights>2002 INIST-CNRS</rights><rights>Springer-Verlag 2001</rights><lds50>peer_reviewed</lds50><woscitedreferencessubscribed>false</woscitedreferencessubscribed><citedby>FETCH-LOGICAL-c346t-b28b4fc7d50d2b599b9fce1504da23845e38724f8a26837090b7519bcd0af23c3</citedby></display><links><openurl>$$Topenurl_article</openurl><openurlfulltext>$$Topenurlfull_article</openurlfulltext><thumbnail>$$Tsyndetics_thumb_exl</thumbnail><link.rule.ids>315,781,785,27929,27930</link.rule.ids><backlink>$$Uhttp://pascal-francis.inist.fr/vibad/index.php?action=getRecordDetail&idt=14138241$$DView record in Pascal Francis$$Hfree_for_read</backlink><backlink>$$Uhttps://www.ncbi.nlm.nih.gov/pubmed/11685305$$D View this record in MEDLINE/PubMed$$Hfree_for_read</backlink></links><search><creatorcontrib>SCHULZE, Andreas</creatorcontrib><creatorcontrib>ABUBAKAR, Kabir</creatorcontrib><creatorcontrib>GILL, Gerald</creatorcontrib><creatorcontrib>WAY, R. Clifton</creatorcontrib><creatorcontrib>SINCLAIR, John C</creatorcontrib><title>Pulmonary oxygen consumption : a hypothesis to explain the increase in oxygen consumption of low birth weight infants with lung disease</title><title>Intensive care medicine</title><addtitle>Intensive Care Med</addtitle><description>We determined pulmonary oxygen consumption (VO2lung) in low-birthweight infants with acute lung disease to help explain the greater whole-body oxygen consumption (VO2wb) in these infants with than in those without lung disease.
Eleven infants (birth weight 1,076+/-364 g; gestational age 28+/-3 weeks) undergoing mechanical ventilation for respiratory distress syndrome were studied in their first week of life. We measured VO2wb by indirect calorimetry and simultaneously determined systemic oxygen uptake (VO2Fick) as the product of cardiac output (echocardiography) and the arterial-mixed venous oxygen content difference (cooximetry) assuming that VO2wb-VO2Fick accounts for VO2lung. Right atrial blood samples were used to determine mixed venous oxygenation, and infants were excluded if samples returned saturations greater than 89%.
VO2lung was 1.92+/-1.74 ml x kg(-1) x min(-1), representing 25% of their VO2wb (7.58+/-1.48 ml x kg(-1) x min(-1)). VO2lung was not correlated with clinical measures of acute disease severity. However, infants with the most severe changes on follow-up radiography (Edwards score 5 as assessed by radiologist blinded for VO2 data) all had a VO2lung level greater than 2.0 ml x kg(-1) x min(-1).
VO2lung can account for the elevated metabolic rate in low-birthweight infants with lung injury. We speculate that this reflects in part inflammatory pulmonary processes and may herald chronic lung disease.</description><subject>Acute Disease</subject><subject>Anesthesia. Intensive care medicine. Transfusions. Cell therapy and gene therapy</subject><subject>Biological and medical sciences</subject><subject>Birth Weight</subject><subject>Calorimetry, Indirect</subject><subject>Carbon Dioxide - blood</subject><subject>Cardiac Output</subject><subject>Chronic Disease</subject><subject>Emergency and intensive respiratory care</subject><subject>Energy Metabolism</subject><subject>Female</subject><subject>Gestational Age</subject><subject>Humans</subject><subject>Hyaline Membrane Disease - diagnostic imaging</subject><subject>Hyaline Membrane Disease - immunology</subject><subject>Hyaline Membrane Disease - metabolism</subject><subject>Hyaline Membrane Disease - physiopathology</subject><subject>Hyaline Membrane Disease - therapy</subject><subject>Hypotheses</subject><subject>Infant, Low Birth Weight</subject><subject>Infant, Newborn</subject><subject>Infant, Premature, Diseases - diagnostic imaging</subject><subject>Infant, Premature, Diseases - immunology</subject><subject>Infant, Premature, Diseases - metabolism</subject><subject>Infant, Premature, Diseases - physiopathology</subject><subject>Infant, Premature, Diseases - therapy</subject><subject>Inflammation</subject><subject>Intensive care medicine</subject><subject>Lung - metabolism</subject><subject>Lung diseases</subject><subject>Lung Diseases - diagnostic imaging</subject><subject>Lung Diseases - immunology</subject><subject>Lung Diseases - metabolism</subject><subject>Lung Diseases - physiopathology</subject><subject>Lung Diseases - therapy</subject><subject>Male</subject><subject>Medical sciences</subject><subject>Oximetry</subject><subject>Oxygen - blood</subject><subject>Oxygen Consumption</subject><subject>Radiography</subject><subject>Respiration, Artificial</subject><subject>Severity of Illness Index</subject><subject>Single-Blind Method</subject><subject>Time Factors</subject><issn>0342-4642</issn><issn>1432-1238</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>2001</creationdate><recordtype>article</recordtype><sourceid>EIF</sourceid><sourceid>ABUWG</sourceid><sourceid>AFKRA</sourceid><sourceid>BENPR</sourceid><sourceid>CCPQU</sourceid><recordid>eNpt0U1rHCEYB3AJDck2yTHXIoX2Nq2vo9NbCH0JBJJDcx4cR3cNMzr1mWGzn6BfOy5ZCC1BUHn4-Ud9ELqk5AslRH0FQigXhJahxBFaUcFZRRnX79CKcMEqUQt2it4DPBapaklP0CmltZacyBX6e78MY4om73B62q1dxDZFWMZpDinib9jgzW5K88ZBADwn7J6mwYSISwWHaLMzsN-8dTh5PKQt7kKeN3jrwnozF-lNnAFvQ6kNS1zjPsA-4xwdezOAuzisZ-jhx_ff17-q27ufN9dXt5Xlop6rjulOeKt6SXrWyabpGm8dlUT0prxZSMe1YsJrw2rNFWlIpyRtOtsT4xm3_Ax9fsmdcvqzOJjbMYB1w2CiSwu0ijHBZKML_PgffExLjuVubdMwrYlQsqDqBdmcALLz7ZTDWD6zpaTdt6f9pz3FfziELt3o-ld96EcBnw7AgDWDzybaAK9OUK5ZmZ4BhTWY6Q</recordid><startdate>20011001</startdate><enddate>20011001</enddate><creator>SCHULZE, Andreas</creator><creator>ABUBAKAR, Kabir</creator><creator>GILL, Gerald</creator><creator>WAY, R. 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Clifton ; SINCLAIR, John C</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-c346t-b28b4fc7d50d2b599b9fce1504da23845e38724f8a26837090b7519bcd0af23c3</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng</language><creationdate>2001</creationdate><topic>Acute Disease</topic><topic>Anesthesia. Intensive care medicine. Transfusions. Cell therapy and gene therapy</topic><topic>Biological and medical sciences</topic><topic>Birth Weight</topic><topic>Calorimetry, Indirect</topic><topic>Carbon Dioxide - blood</topic><topic>Cardiac Output</topic><topic>Chronic Disease</topic><topic>Emergency and intensive respiratory care</topic><topic>Energy Metabolism</topic><topic>Female</topic><topic>Gestational Age</topic><topic>Humans</topic><topic>Hyaline Membrane Disease - diagnostic imaging</topic><topic>Hyaline Membrane Disease - immunology</topic><topic>Hyaline Membrane Disease - metabolism</topic><topic>Hyaline Membrane Disease - physiopathology</topic><topic>Hyaline Membrane Disease - therapy</topic><topic>Hypotheses</topic><topic>Infant, Low Birth Weight</topic><topic>Infant, Newborn</topic><topic>Infant, Premature, Diseases - diagnostic imaging</topic><topic>Infant, Premature, Diseases - immunology</topic><topic>Infant, Premature, Diseases - metabolism</topic><topic>Infant, Premature, Diseases - physiopathology</topic><topic>Infant, Premature, Diseases - therapy</topic><topic>Inflammation</topic><topic>Intensive care medicine</topic><topic>Lung - metabolism</topic><topic>Lung diseases</topic><topic>Lung Diseases - diagnostic imaging</topic><topic>Lung Diseases - immunology</topic><topic>Lung Diseases - metabolism</topic><topic>Lung Diseases - physiopathology</topic><topic>Lung Diseases - therapy</topic><topic>Male</topic><topic>Medical sciences</topic><topic>Oximetry</topic><topic>Oxygen - blood</topic><topic>Oxygen Consumption</topic><topic>Radiography</topic><topic>Respiration, Artificial</topic><topic>Severity of Illness Index</topic><topic>Single-Blind Method</topic><topic>Time Factors</topic><toplevel>peer_reviewed</toplevel><toplevel>online_resources</toplevel><creatorcontrib>SCHULZE, Andreas</creatorcontrib><creatorcontrib>ABUBAKAR, Kabir</creatorcontrib><creatorcontrib>GILL, Gerald</creatorcontrib><creatorcontrib>WAY, R. 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Clifton</au><au>SINCLAIR, John C</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>Pulmonary oxygen consumption : a hypothesis to explain the increase in oxygen consumption of low birth weight infants with lung disease</atitle><jtitle>Intensive care medicine</jtitle><addtitle>Intensive Care Med</addtitle><date>2001-10-01</date><risdate>2001</risdate><volume>27</volume><issue>10</issue><spage>1636</spage><epage>1642</epage><pages>1636-1642</pages><issn>0342-4642</issn><eissn>1432-1238</eissn><coden>ICMED9</coden><abstract>We determined pulmonary oxygen consumption (VO2lung) in low-birthweight infants with acute lung disease to help explain the greater whole-body oxygen consumption (VO2wb) in these infants with than in those without lung disease.
Eleven infants (birth weight 1,076+/-364 g; gestational age 28+/-3 weeks) undergoing mechanical ventilation for respiratory distress syndrome were studied in their first week of life. We measured VO2wb by indirect calorimetry and simultaneously determined systemic oxygen uptake (VO2Fick) as the product of cardiac output (echocardiography) and the arterial-mixed venous oxygen content difference (cooximetry) assuming that VO2wb-VO2Fick accounts for VO2lung. Right atrial blood samples were used to determine mixed venous oxygenation, and infants were excluded if samples returned saturations greater than 89%.
VO2lung was 1.92+/-1.74 ml x kg(-1) x min(-1), representing 25% of their VO2wb (7.58+/-1.48 ml x kg(-1) x min(-1)). VO2lung was not correlated with clinical measures of acute disease severity. However, infants with the most severe changes on follow-up radiography (Edwards score 5 as assessed by radiologist blinded for VO2 data) all had a VO2lung level greater than 2.0 ml x kg(-1) x min(-1).
VO2lung can account for the elevated metabolic rate in low-birthweight infants with lung injury. We speculate that this reflects in part inflammatory pulmonary processes and may herald chronic lung disease.</abstract><cop>Heidelberg</cop><cop>Berlin</cop><pub>Springer</pub><pmid>11685305</pmid><doi>10.1007/s001340101074</doi><tpages>7</tpages></addata></record> |
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| subjects | Acute Disease Anesthesia. Intensive care medicine. Transfusions. Cell therapy and gene therapy Biological and medical sciences Birth Weight Calorimetry, Indirect Carbon Dioxide - blood Cardiac Output Chronic Disease Emergency and intensive respiratory care Energy Metabolism Female Gestational Age Humans Hyaline Membrane Disease - diagnostic imaging Hyaline Membrane Disease - immunology Hyaline Membrane Disease - metabolism Hyaline Membrane Disease - physiopathology Hyaline Membrane Disease - therapy Hypotheses Infant, Low Birth Weight Infant, Newborn Infant, Premature, Diseases - diagnostic imaging Infant, Premature, Diseases - immunology Infant, Premature, Diseases - metabolism Infant, Premature, Diseases - physiopathology Infant, Premature, Diseases - therapy Inflammation Intensive care medicine Lung - metabolism Lung diseases Lung Diseases - diagnostic imaging Lung Diseases - immunology Lung Diseases - metabolism Lung Diseases - physiopathology Lung Diseases - therapy Male Medical sciences Oximetry Oxygen - blood Oxygen Consumption Radiography Respiration, Artificial Severity of Illness Index Single-Blind Method Time Factors |
| title | Pulmonary oxygen consumption : a hypothesis to explain the increase in oxygen consumption of low birth weight infants with lung disease |
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