Acute Gastric Dilatation Revisited
First described by S.E. Duplay in 1833, acute gastric dilatation has since been well documented in the literature. Several theories of the pathogenesis of acute gastric dilatation have been postulated. In 1842, Karl Freiherr von Rokitansky described the superior mesenteric artery syndrome, followed...
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Veröffentlicht in: | The American surgeon 2000-08, Vol.66 (8), p.709-710 |
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description | First described by S.E. Duplay in 1833, acute gastric dilatation has since been well documented in the literature. Several theories of the pathogenesis of acute gastric dilatation have been postulated. In 1842, Karl Freiherr von Rokitansky described the superior mesenteric artery syndrome, followed by W. Brinton in 1859 with the atonic theory. C.R. Morris et al. introduced debilitation and anesthesia as predisposing factors. Although rare, gastric necrosis is the most severe consequence of acute gastric dilatation. Vascular insufficiency secondary to increased intragastric pressure is the critical factor. We report an unusual case of acute gastric dilatation with subsequent necrosis of uncertain etiology. |
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Rob ; Marshall, Gary T. ; Tyroch, Alan H.</creator><creatorcontrib>Todd, S. Rob ; Marshall, Gary T. ; Tyroch, Alan H.</creatorcontrib><description>First described by S.E. Duplay in 1833, acute gastric dilatation has since been well documented in the literature. Several theories of the pathogenesis of acute gastric dilatation have been postulated. In 1842, Karl Freiherr von Rokitansky described the superior mesenteric artery syndrome, followed by W. Brinton in 1859 with the atonic theory. C.R. Morris et al. introduced debilitation and anesthesia as predisposing factors. Although rare, gastric necrosis is the most severe consequence of acute gastric dilatation. Vascular insufficiency secondary to increased intragastric pressure is the critical factor. 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Rob</creatorcontrib><creatorcontrib>Marshall, Gary T.</creatorcontrib><creatorcontrib>Tyroch, Alan H.</creatorcontrib><title>Acute Gastric Dilatation Revisited</title><title>The American surgeon</title><addtitle>Am Surg</addtitle><description>First described by S.E. Duplay in 1833, acute gastric dilatation has since been well documented in the literature. Several theories of the pathogenesis of acute gastric dilatation have been postulated. In 1842, Karl Freiherr von Rokitansky described the superior mesenteric artery syndrome, followed by W. Brinton in 1859 with the atonic theory. C.R. Morris et al. introduced debilitation and anesthesia as predisposing factors. Although rare, gastric necrosis is the most severe consequence of acute gastric dilatation. Vascular insufficiency secondary to increased intragastric pressure is the critical factor. We report an unusual case of acute gastric dilatation with subsequent necrosis of uncertain etiology.</description><subject>Acute Disease</subject><subject>Aged</subject><subject>Biological and medical sciences</subject><subject>Disease Progression</subject><subject>Fatal Outcome</subject><subject>Gastric Dilatation - etiology</subject><subject>Gastric Dilatation - pathology</subject><subject>Gastric Dilatation - surgery</subject><subject>Gastric Dilatation - therapy</subject><subject>Gastroenterology. Liver. Pancreas. Abdomen</subject><subject>Humans</subject><subject>Illnesses</subject><subject>Male</subject><subject>Medical sciences</subject><subject>Other diseases. Semiology</subject><subject>Stomach</subject><subject>Stomach - pathology</subject><subject>Stomach. Duodenum. Small intestine. Colon. Rectum. Anus</subject><subject>Vomiting - etiology</subject><issn>0003-1348</issn><issn>1555-9823</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>2000</creationdate><recordtype>article</recordtype><sourceid>EIF</sourceid><sourceid>8G5</sourceid><sourceid>ABUWG</sourceid><sourceid>AFKRA</sourceid><sourceid>AZQEC</sourceid><sourceid>BENPR</sourceid><sourceid>CCPQU</sourceid><sourceid>DWQXO</sourceid><sourceid>GNUQQ</sourceid><sourceid>GUQSH</sourceid><sourceid>M2O</sourceid><recordid>eNp90E1LAzEQBuAgiq3VP-BBShFvazP52uRYqlahIIiel2w2kZRttya7gv_elC1UFMxlCDwzeTMIXQK-BcjzKcaYAmUyVSEwlhiO0BA455mShB6j4Q5kOzFAZzGu0pUJDqdoAFilDkKGaDIzXWvHCx3b4M34zte61a1vNuMX--mjb211jk6crqO92NcRenu4f50_ZsvnxdN8tswME9BmZSWwcBVVzJpKKScYcJkTSUE5ZimvNFeqrHKbUymVLbFzuEzJtRLUAVN0hG76udvQfHQ2tsXaR2PrWm9s08UiJwTSf0WCk19w1XRhk7IVBIjEifCESI9MaGIM1hXb4Nc6fBWAi936ir_rS01X-8ldubbVj5Z-Xwlc74GORtcu6I3x8eAYxzSdEZr2LOp3e0j3z8vfeKWAsg</recordid><startdate>20000801</startdate><enddate>20000801</enddate><creator>Todd, S. 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Rob ; Marshall, Gary T. ; Tyroch, Alan H.</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-c461t-bd606fd394ecd99f64158728319f4e35da599bd7e73889eb0ff0b006a963f1493</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng</language><creationdate>2000</creationdate><topic>Acute Disease</topic><topic>Aged</topic><topic>Biological and medical sciences</topic><topic>Disease Progression</topic><topic>Fatal Outcome</topic><topic>Gastric Dilatation - etiology</topic><topic>Gastric Dilatation - pathology</topic><topic>Gastric Dilatation - surgery</topic><topic>Gastric Dilatation - therapy</topic><topic>Gastroenterology. Liver. Pancreas. Abdomen</topic><topic>Humans</topic><topic>Illnesses</topic><topic>Male</topic><topic>Medical sciences</topic><topic>Other diseases. Semiology</topic><topic>Stomach</topic><topic>Stomach - pathology</topic><topic>Stomach. Duodenum. Small intestine. Colon. Rectum. 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Rob</au><au>Marshall, Gary T.</au><au>Tyroch, Alan H.</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>Acute Gastric Dilatation Revisited</atitle><jtitle>The American surgeon</jtitle><addtitle>Am Surg</addtitle><date>2000-08-01</date><risdate>2000</risdate><volume>66</volume><issue>8</issue><spage>709</spage><epage>710</epage><pages>709-710</pages><issn>0003-1348</issn><eissn>1555-9823</eissn><coden>AMSUAW</coden><abstract>First described by S.E. Duplay in 1833, acute gastric dilatation has since been well documented in the literature. Several theories of the pathogenesis of acute gastric dilatation have been postulated. In 1842, Karl Freiherr von Rokitansky described the superior mesenteric artery syndrome, followed by W. Brinton in 1859 with the atonic theory. C.R. Morris et al. introduced debilitation and anesthesia as predisposing factors. Although rare, gastric necrosis is the most severe consequence of acute gastric dilatation. Vascular insufficiency secondary to increased intragastric pressure is the critical factor. We report an unusual case of acute gastric dilatation with subsequent necrosis of uncertain etiology.</abstract><cop>Los Angeles, CA</cop><pub>SAGE Publications</pub><pmid>10966022</pmid><doi>10.1177/000313480006600801</doi><tpages>2</tpages></addata></record> |
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subjects | Acute Disease Aged Biological and medical sciences Disease Progression Fatal Outcome Gastric Dilatation - etiology Gastric Dilatation - pathology Gastric Dilatation - surgery Gastric Dilatation - therapy Gastroenterology. Liver. Pancreas. Abdomen Humans Illnesses Male Medical sciences Other diseases. Semiology Stomach Stomach - pathology Stomach. Duodenum. Small intestine. Colon. Rectum. Anus Vomiting - etiology |
title | Acute Gastric Dilatation Revisited |
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