Increased AT(1) receptors in adrenal gland of AT(2) receptor gene-disrupted mice

Angiotensin II (Ang II) AT(2) receptor-gene disrupted mice have increased systemic blood pressure and response to exogenous Angiotensin II. To clarify the mechanism of these changes, we studied adrenal AT(1) receptor expression and mRNA by receptor autoradiography and in situ hybridization in female...

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Veröffentlicht in:	Regulatory peptides 2001-10, Vol.102 (1), p.41-47
Hauptverfasser:	Saavedra, J M, Armando, I, Terrón, J A, Falcón-Neri, A, Jöhren, O, Häuser, W, Inagami, T
Format:	Artikel
Sprache:	eng
Schlagworte:	Adrenal Medulla - metabolism Angiotensin II - pharmacology Animals Autoradiography Blood Pressure - drug effects Female Gene Expression Regulation Genotype Imidazoles - pharmacology In Situ Hybridization Losartan - pharmacology Mice Mice, Inbred Strains Mice, Knockout Mice, Mutant Strains Pyridines - pharmacology Receptor, Angiotensin, Type 1 Receptor, Angiotensin, Type 2 Receptors, Angiotensin - deficiency Receptors, Angiotensin - genetics Receptors, Angiotensin - metabolism RNA, Messenger - metabolism Transcription, Genetic Vasoconstrictor Agents - pharmacology Zona Glomerulosa - drug effects Zona Glomerulosa - metabolism
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