Venous thrombosis and tissue plasminogen activator release deficiency: a family study

Thrombotic events are often due to fibrinolytic defects such as impaired tissue plasminogen activator (tPA) synthesis and/or release or increased plasminogen activator inhibitor (PAI) levels. In this report we describe four members of a family with a history of recurrent venous thrombosis, who demon...

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Veröffentlicht in:	Blood coagulation & fibrinolysis 1991-04, Vol.2 (2), p.231-236
Hauptverfasser:	Pattassi, G M, Sartori, M T, Viero, M L, Boscaro, M, Boeri, G, Girolami, A
Format:	Artikel
Sprache:	eng
Schlagworte:	Adult Constriction Deamino Arginine Vasopressin Female Fibrinolysis Humans Male Pedigree Plasminogen - metabolism Thrombophlebitis - etiology Thrombophlebitis - genetics Tissue Plasminogen Activator - deficiency Tissue Plasminogen Activator - metabolism Veins
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