Role of induced nitric oxide in the initiation of the inflammatory response after postischemic injury

The aim of this study was to investigate the role of inducible nitric oxide (NO) synthase (iNOS) and NO on the modulation of the inflammatory response caused by splanchnic ischemia and reperfusion. A severe model of mesenteric ischemia and reperfusion was produced by subjecting mice to 45 min occlus...

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Veröffentlicht in:	Shock (Augusta, Ga.) Ga.), 2002-08, Vol.18 (2), p.169-176
Hauptverfasser:	CUZZOCREA, Salvatore, CHATTERJEE, Prabal K, MAZZON, Emanuela, DUGO, Laura, DE SARRO, Angela, VAN DE LOO, Fons A. J, CAPUTI, Achille P, THIEMERMANN, Christoph
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Biological and medical sciences Cytokines - analysis Cytokines - metabolism Disease Models, Animal Gastroenterology. Liver. Pancreas. Abdomen Immunohistochemistry Inflammation Mediators - analysis Ischemia - metabolism Ischemia - mortality Ischemia - pathology Lipid Peroxidation Male Malondialdehyde - analysis Medical sciences Mesenteric Arteries Mesenteric Vascular Occlusion - metabolism Mesenteric Vascular Occlusion - mortality Mesenteric Vascular Occlusion - pathology Mice Mice, Knockout Neutrophil Infiltration Nitric Oxide - analysis Nitric Oxide - metabolism Nitric Oxide Synthase - analysis Nitric Oxide Synthase - metabolism Other diseases. Semiology P-Selectin - analysis Peroxidase - analysis Reperfusion Injury - metabolism Reperfusion Injury - physiopathology Sensitivity and Specificity Stomach. Duodenum. Small intestine. Colon. Rectum. Anus Survival Rate
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