Angiotensin II receptors from peritransplantation through first-year post-transplantation and the risk of transplant coronary artery disease
We evaluated whether the angiotensin II (Ang II) receptors from perioperation through one-year post-transplantation predict the transplant coronary artery disease (TCAD) progression. The role of Ang II receptors (type 1: AT1R; type 2: AT2R) in TCAD is uncertain. We investigated 28 heart donors and t...
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| Veröffentlicht in: | Journal of the American College of Cardiology 2004-05, Vol.43 (9), p.1565-1573 |
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| creator | Yousufuddin, Mohammed Cook, Daniel J Starling, Randall C Abdo, Ashraf Paul, Philip Tuzcu, E.Murat Ratliff, Norman B McCarthy, Patrick M Young, James B Yamani, Mohamad H |
| description | We evaluated whether the angiotensin II (Ang II) receptors from perioperation through one-year post-transplantation predict the transplant coronary artery disease (TCAD) progression.
The role of Ang II receptors (type 1: AT1R; type 2: AT2R) in TCAD is uncertain.
We investigated 28 heart donors and the corresponding recipients. The levels of AT1R and AT2R messenger ribonucleic acid (mRNA) were examined in lymphocytes from the donor spleen and in the donor heart at one-week and one-year posttransplantation to determine their association with the progression of TCAD, measured as changes in maximal intimal thickness (CMIT) and plaque volume (CPV) by intravascular ultrasound (IVUS) examinations.
The AT1R mRNA in lymphocytes from the donor spleen (CMIT: r = 0.73, p < 0.0001; CPV: r = 0.69, p < 0.0001) and in the donor hearts at one-week (CMIT: r = 0.52, p = 0.005; CPV: r = 0.56, p = 0.002) and at one-year (CMIT: r = 0.63, p < 0.0001; CPV: r = 0.43, p = 0.004) post-transplantation along with AT2R mRNA in the donor hearts at one-year post-transplantation (CMIT: r = 0.3, p < 0.0001; CPV: r = 0.53, p = 0.009) were univariate predictors, whereas AT1R mRNA in lymphocytes and in the donor hearts at one-year post-transplantation proved to be multivariate predictors of the progression of TCAD.
These data suggest a role for Ang II receptors in the pathogenesis of TCAD and support a novel concept that TCAD may have its origin in the donor per se and may be modulated by the recipient's inherent biological factors. |
| doi_str_mv | 10.1016/j.jacc.2003.11.060 |
| format | Article |
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The role of Ang II receptors (type 1: AT1R; type 2: AT2R) in TCAD is uncertain.
We investigated 28 heart donors and the corresponding recipients. The levels of AT1R and AT2R messenger ribonucleic acid (mRNA) were examined in lymphocytes from the donor spleen and in the donor heart at one-week and one-year posttransplantation to determine their association with the progression of TCAD, measured as changes in maximal intimal thickness (CMIT) and plaque volume (CPV) by intravascular ultrasound (IVUS) examinations.
The AT1R mRNA in lymphocytes from the donor spleen (CMIT: r = 0.73, p < 0.0001; CPV: r = 0.69, p < 0.0001) and in the donor hearts at one-week (CMIT: r = 0.52, p = 0.005; CPV: r = 0.56, p = 0.002) and at one-year (CMIT: r = 0.63, p < 0.0001; CPV: r = 0.43, p = 0.004) post-transplantation along with AT2R mRNA in the donor hearts at one-year post-transplantation (CMIT: r = 0.3, p < 0.0001; CPV: r = 0.53, p = 0.009) were univariate predictors, whereas AT1R mRNA in lymphocytes and in the donor hearts at one-year post-transplantation proved to be multivariate predictors of the progression of TCAD.
These data suggest a role for Ang II receptors in the pathogenesis of TCAD and support a novel concept that TCAD may have its origin in the donor per se and may be modulated by the recipient's inherent biological factors.</description><identifier>ISSN: 0735-1097</identifier><identifier>EISSN: 1558-3597</identifier><identifier>DOI: 10.1016/j.jacc.2003.11.060</identifier><identifier>PMID: 15120813</identifier><identifier>CODEN: JACCDI</identifier><language>eng</language><publisher>New York, NY: Elsevier Inc</publisher><subject>Adult ; Aged ; Angiotensin-Converting Enzyme Inhibitors - therapeutic use ; Biological and medical sciences ; Biopsy ; Cardiology ; Cardiology. Vascular system ; Cardiovascular disease ; Coronary Artery Disease - epidemiology ; Coronary Artery Disease - etiology ; Coronary Artery Disease - metabolism ; Coronary heart disease ; Coronary Vessels - diagnostic imaging ; Coronary Vessels - metabolism ; Coronary Vessels - pathology ; Cytomegalovirus ; Disease Progression ; Female ; Fluorescent Antibody Technique ; Follow-Up Studies ; Heart ; Heart Failure - drug therapy ; Heart Failure - metabolism ; Heart Failure - surgery ; Heart Transplantation ; Humans ; Lymphocytes ; Lymphocytes - metabolism ; Male ; Medical sciences ; Middle Aged ; Mortality ; Multivariate Analysis ; Pathogenesis ; Postoperative Complications - epidemiology ; Postoperative Complications - etiology ; Postoperative Complications - metabolism ; Predictive Value of Tests ; Receptor, Angiotensin, Type 1 - biosynthesis ; Receptors, Angiotensin - biosynthesis ; Risk Factors ; RNA, Messenger - metabolism ; Spleen ; Studies ; Tissue Donors ; Transplants & implants ; Treatment Outcome ; Tunica Intima - diagnostic imaging ; Tunica Intima - metabolism ; Tunica Intima - pathology ; Ultrasonography, Interventional</subject><ispartof>Journal of the American College of Cardiology, 2004-05, Vol.43 (9), p.1565-1573</ispartof><rights>2004 American College of Cardiology Foundation</rights><rights>2004 INIST-CNRS</rights><rights>Copyright Elsevier Limited May 5, 2004</rights><lds50>peer_reviewed</lds50><oa>free_for_read</oa><woscitedreferencessubscribed>false</woscitedreferencessubscribed><citedby>FETCH-LOGICAL-c445t-c9754f8a1cf28d624895e6861bb7dc0460e0029e3ea89513fcc50fde37496e9e3</citedby><cites>FETCH-LOGICAL-c445t-c9754f8a1cf28d624895e6861bb7dc0460e0029e3ea89513fcc50fde37496e9e3</cites></display><links><openurl>$$Topenurl_article</openurl><openurlfulltext>$$Topenurlfull_article</openurlfulltext><thumbnail>$$Tsyndetics_thumb_exl</thumbnail><linktohtml>$$Uhttps://www.sciencedirect.com/science/article/pii/S0735109704003596$$EHTML$$P50$$Gelsevier$$Hfree_for_read</linktohtml><link.rule.ids>314,776,780,3537,27901,27902,65306</link.rule.ids><backlink>$$Uhttp://pascal-francis.inist.fr/vibad/index.php?action=getRecordDetail&idt=15725477$$DView record in Pascal Francis$$Hfree_for_read</backlink><backlink>$$Uhttps://www.ncbi.nlm.nih.gov/pubmed/15120813$$D View this record in MEDLINE/PubMed$$Hfree_for_read</backlink></links><search><creatorcontrib>Yousufuddin, Mohammed</creatorcontrib><creatorcontrib>Cook, Daniel J</creatorcontrib><creatorcontrib>Starling, Randall C</creatorcontrib><creatorcontrib>Abdo, Ashraf</creatorcontrib><creatorcontrib>Paul, Philip</creatorcontrib><creatorcontrib>Tuzcu, E.Murat</creatorcontrib><creatorcontrib>Ratliff, Norman B</creatorcontrib><creatorcontrib>McCarthy, Patrick M</creatorcontrib><creatorcontrib>Young, James B</creatorcontrib><creatorcontrib>Yamani, Mohamad H</creatorcontrib><title>Angiotensin II receptors from peritransplantation through first-year post-transplantation and the risk of transplant coronary artery disease</title><title>Journal of the American College of Cardiology</title><addtitle>J Am Coll Cardiol</addtitle><description>We evaluated whether the angiotensin II (Ang II) receptors from perioperation through one-year post-transplantation predict the transplant coronary artery disease (TCAD) progression.
The role of Ang II receptors (type 1: AT1R; type 2: AT2R) in TCAD is uncertain.
We investigated 28 heart donors and the corresponding recipients. The levels of AT1R and AT2R messenger ribonucleic acid (mRNA) were examined in lymphocytes from the donor spleen and in the donor heart at one-week and one-year posttransplantation to determine their association with the progression of TCAD, measured as changes in maximal intimal thickness (CMIT) and plaque volume (CPV) by intravascular ultrasound (IVUS) examinations.
The AT1R mRNA in lymphocytes from the donor spleen (CMIT: r = 0.73, p < 0.0001; CPV: r = 0.69, p < 0.0001) and in the donor hearts at one-week (CMIT: r = 0.52, p = 0.005; CPV: r = 0.56, p = 0.002) and at one-year (CMIT: r = 0.63, p < 0.0001; CPV: r = 0.43, p = 0.004) post-transplantation along with AT2R mRNA in the donor hearts at one-year post-transplantation (CMIT: r = 0.3, p < 0.0001; CPV: r = 0.53, p = 0.009) were univariate predictors, whereas AT1R mRNA in lymphocytes and in the donor hearts at one-year post-transplantation proved to be multivariate predictors of the progression of TCAD.
These data suggest a role for Ang II receptors in the pathogenesis of TCAD and support a novel concept that TCAD may have its origin in the donor per se and may be modulated by the recipient's inherent biological factors.</description><subject>Adult</subject><subject>Aged</subject><subject>Angiotensin-Converting Enzyme Inhibitors - therapeutic use</subject><subject>Biological and medical sciences</subject><subject>Biopsy</subject><subject>Cardiology</subject><subject>Cardiology. Vascular system</subject><subject>Cardiovascular disease</subject><subject>Coronary Artery Disease - epidemiology</subject><subject>Coronary Artery Disease - etiology</subject><subject>Coronary Artery Disease - metabolism</subject><subject>Coronary heart disease</subject><subject>Coronary Vessels - diagnostic imaging</subject><subject>Coronary Vessels - metabolism</subject><subject>Coronary Vessels - pathology</subject><subject>Cytomegalovirus</subject><subject>Disease Progression</subject><subject>Female</subject><subject>Fluorescent Antibody Technique</subject><subject>Follow-Up Studies</subject><subject>Heart</subject><subject>Heart Failure - drug therapy</subject><subject>Heart Failure - metabolism</subject><subject>Heart Failure - surgery</subject><subject>Heart Transplantation</subject><subject>Humans</subject><subject>Lymphocytes</subject><subject>Lymphocytes - metabolism</subject><subject>Male</subject><subject>Medical sciences</subject><subject>Middle Aged</subject><subject>Mortality</subject><subject>Multivariate Analysis</subject><subject>Pathogenesis</subject><subject>Postoperative Complications - epidemiology</subject><subject>Postoperative Complications - etiology</subject><subject>Postoperative Complications - metabolism</subject><subject>Predictive Value of Tests</subject><subject>Receptor, Angiotensin, Type 1 - biosynthesis</subject><subject>Receptors, Angiotensin - biosynthesis</subject><subject>Risk Factors</subject><subject>RNA, Messenger - metabolism</subject><subject>Spleen</subject><subject>Studies</subject><subject>Tissue Donors</subject><subject>Transplants & implants</subject><subject>Treatment Outcome</subject><subject>Tunica Intima - diagnostic imaging</subject><subject>Tunica Intima - metabolism</subject><subject>Tunica Intima - pathology</subject><subject>Ultrasonography, Interventional</subject><issn>0735-1097</issn><issn>1558-3597</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>2004</creationdate><recordtype>article</recordtype><sourceid>EIF</sourceid><recordid>eNp9kc2KFDEQgIMo7rj6Ah4kIHrrtqq70-kGL8viz8CCFz2HTLqym3YmaZO0sO_gQ5thBlb24KlC6quiqj7GXiPUCNh_mOtZG1M3AG2NWEMPT9gGhRiqVozyKduAbEWFMMoL9iKlGQD6Acfn7AIFNjBgu2F_rvytC5l8cp5vtzySoSWHmLiN4cAXii5H7dOy1z7r7ILn-S6G9faOWxdTru5JR76E8nrMaT8Vlnh06ScPlj_kuQkxeB3vuY6ZSphcIp3oJXtm9T7Rq3O8ZD8-f_p-_bW6-fZle311U5muE7kyoxSdHTQa2wxT33TDKKgfetzt5GSg64EAmpFa0iWDrTVGgJ2old3YU_m_ZO9PfZcYfq2Usjq4ZGhfZqOwJiVxGLsWmwK-fQTOYY2-zKZQQI-dHOVQqOZEmRhSimTVEt2hrKcQ1NGUmtXRlDqaUoiqmCpFb86t192BpoeSs5oCvDsDOhm9t-V6xqV_ONmITsrCfTxxVC7221FUyTjyhiZXZGY1Bfe_Of4CLIi0lg</recordid><startdate>20040505</startdate><enddate>20040505</enddate><creator>Yousufuddin, Mohammed</creator><creator>Cook, Daniel J</creator><creator>Starling, Randall C</creator><creator>Abdo, Ashraf</creator><creator>Paul, Philip</creator><creator>Tuzcu, E.Murat</creator><creator>Ratliff, Norman B</creator><creator>McCarthy, Patrick M</creator><creator>Young, James B</creator><creator>Yamani, Mohamad H</creator><general>Elsevier Inc</general><general>Elsevier Science</general><general>Elsevier Limited</general><scope>6I.</scope><scope>AAFTH</scope><scope>IQODW</scope><scope>CGR</scope><scope>CUY</scope><scope>CVF</scope><scope>ECM</scope><scope>EIF</scope><scope>NPM</scope><scope>AAYXX</scope><scope>CITATION</scope><scope>7T5</scope><scope>7TK</scope><scope>H94</scope><scope>K9.</scope><scope>NAPCQ</scope><scope>7X8</scope></search><sort><creationdate>20040505</creationdate><title>Angiotensin II receptors from peritransplantation through first-year post-transplantation and the risk of transplant coronary artery disease</title><author>Yousufuddin, Mohammed ; Cook, Daniel J ; Starling, Randall C ; Abdo, Ashraf ; Paul, Philip ; Tuzcu, E.Murat ; Ratliff, Norman B ; McCarthy, Patrick M ; Young, James B ; Yamani, Mohamad H</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-c445t-c9754f8a1cf28d624895e6861bb7dc0460e0029e3ea89513fcc50fde37496e9e3</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng</language><creationdate>2004</creationdate><topic>Adult</topic><topic>Aged</topic><topic>Angiotensin-Converting Enzyme Inhibitors - therapeutic use</topic><topic>Biological and medical sciences</topic><topic>Biopsy</topic><topic>Cardiology</topic><topic>Cardiology. Vascular system</topic><topic>Cardiovascular disease</topic><topic>Coronary Artery Disease - epidemiology</topic><topic>Coronary Artery Disease - etiology</topic><topic>Coronary Artery Disease - metabolism</topic><topic>Coronary heart disease</topic><topic>Coronary Vessels - diagnostic imaging</topic><topic>Coronary Vessels - metabolism</topic><topic>Coronary Vessels - pathology</topic><topic>Cytomegalovirus</topic><topic>Disease Progression</topic><topic>Female</topic><topic>Fluorescent Antibody Technique</topic><topic>Follow-Up Studies</topic><topic>Heart</topic><topic>Heart Failure - drug therapy</topic><topic>Heart Failure - metabolism</topic><topic>Heart Failure - surgery</topic><topic>Heart Transplantation</topic><topic>Humans</topic><topic>Lymphocytes</topic><topic>Lymphocytes - metabolism</topic><topic>Male</topic><topic>Medical sciences</topic><topic>Middle Aged</topic><topic>Mortality</topic><topic>Multivariate Analysis</topic><topic>Pathogenesis</topic><topic>Postoperative Complications - epidemiology</topic><topic>Postoperative Complications - etiology</topic><topic>Postoperative Complications - metabolism</topic><topic>Predictive Value of Tests</topic><topic>Receptor, Angiotensin, Type 1 - biosynthesis</topic><topic>Receptors, Angiotensin - biosynthesis</topic><topic>Risk Factors</topic><topic>RNA, Messenger - metabolism</topic><topic>Spleen</topic><topic>Studies</topic><topic>Tissue Donors</topic><topic>Transplants & implants</topic><topic>Treatment Outcome</topic><topic>Tunica Intima - diagnostic imaging</topic><topic>Tunica Intima - metabolism</topic><topic>Tunica Intima - pathology</topic><topic>Ultrasonography, Interventional</topic><toplevel>peer_reviewed</toplevel><toplevel>online_resources</toplevel><creatorcontrib>Yousufuddin, Mohammed</creatorcontrib><creatorcontrib>Cook, Daniel J</creatorcontrib><creatorcontrib>Starling, Randall C</creatorcontrib><creatorcontrib>Abdo, Ashraf</creatorcontrib><creatorcontrib>Paul, Philip</creatorcontrib><creatorcontrib>Tuzcu, E.Murat</creatorcontrib><creatorcontrib>Ratliff, Norman B</creatorcontrib><creatorcontrib>McCarthy, Patrick M</creatorcontrib><creatorcontrib>Young, James B</creatorcontrib><creatorcontrib>Yamani, Mohamad H</creatorcontrib><collection>ScienceDirect Open Access Titles</collection><collection>Elsevier:ScienceDirect:Open Access</collection><collection>Pascal-Francis</collection><collection>Medline</collection><collection>MEDLINE</collection><collection>MEDLINE (Ovid)</collection><collection>MEDLINE</collection><collection>MEDLINE</collection><collection>PubMed</collection><collection>CrossRef</collection><collection>Immunology Abstracts</collection><collection>Neurosciences Abstracts</collection><collection>AIDS and Cancer Research Abstracts</collection><collection>ProQuest Health & Medical Complete (Alumni)</collection><collection>Nursing & Allied Health Premium</collection><collection>MEDLINE - Academic</collection><jtitle>Journal of the American College of Cardiology</jtitle></facets><delivery><delcategory>Remote Search Resource</delcategory><fulltext>fulltext</fulltext></delivery><addata><au>Yousufuddin, Mohammed</au><au>Cook, Daniel J</au><au>Starling, Randall C</au><au>Abdo, Ashraf</au><au>Paul, Philip</au><au>Tuzcu, E.Murat</au><au>Ratliff, Norman B</au><au>McCarthy, Patrick M</au><au>Young, James B</au><au>Yamani, Mohamad H</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>Angiotensin II receptors from peritransplantation through first-year post-transplantation and the risk of transplant coronary artery disease</atitle><jtitle>Journal of the American College of Cardiology</jtitle><addtitle>J Am Coll Cardiol</addtitle><date>2004-05-05</date><risdate>2004</risdate><volume>43</volume><issue>9</issue><spage>1565</spage><epage>1573</epage><pages>1565-1573</pages><issn>0735-1097</issn><eissn>1558-3597</eissn><coden>JACCDI</coden><abstract>We evaluated whether the angiotensin II (Ang II) receptors from perioperation through one-year post-transplantation predict the transplant coronary artery disease (TCAD) progression.
The role of Ang II receptors (type 1: AT1R; type 2: AT2R) in TCAD is uncertain.
We investigated 28 heart donors and the corresponding recipients. The levels of AT1R and AT2R messenger ribonucleic acid (mRNA) were examined in lymphocytes from the donor spleen and in the donor heart at one-week and one-year posttransplantation to determine their association with the progression of TCAD, measured as changes in maximal intimal thickness (CMIT) and plaque volume (CPV) by intravascular ultrasound (IVUS) examinations.
The AT1R mRNA in lymphocytes from the donor spleen (CMIT: r = 0.73, p < 0.0001; CPV: r = 0.69, p < 0.0001) and in the donor hearts at one-week (CMIT: r = 0.52, p = 0.005; CPV: r = 0.56, p = 0.002) and at one-year (CMIT: r = 0.63, p < 0.0001; CPV: r = 0.43, p = 0.004) post-transplantation along with AT2R mRNA in the donor hearts at one-year post-transplantation (CMIT: r = 0.3, p < 0.0001; CPV: r = 0.53, p = 0.009) were univariate predictors, whereas AT1R mRNA in lymphocytes and in the donor hearts at one-year post-transplantation proved to be multivariate predictors of the progression of TCAD.
These data suggest a role for Ang II receptors in the pathogenesis of TCAD and support a novel concept that TCAD may have its origin in the donor per se and may be modulated by the recipient's inherent biological factors.</abstract><cop>New York, NY</cop><pub>Elsevier Inc</pub><pmid>15120813</pmid><doi>10.1016/j.jacc.2003.11.060</doi><tpages>9</tpages><oa>free_for_read</oa></addata></record> |
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| ispartof | Journal of the American College of Cardiology, 2004-05, Vol.43 (9), p.1565-1573 |
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| subjects | Adult Aged Angiotensin-Converting Enzyme Inhibitors - therapeutic use Biological and medical sciences Biopsy Cardiology Cardiology. Vascular system Cardiovascular disease Coronary Artery Disease - epidemiology Coronary Artery Disease - etiology Coronary Artery Disease - metabolism Coronary heart disease Coronary Vessels - diagnostic imaging Coronary Vessels - metabolism Coronary Vessels - pathology Cytomegalovirus Disease Progression Female Fluorescent Antibody Technique Follow-Up Studies Heart Heart Failure - drug therapy Heart Failure - metabolism Heart Failure - surgery Heart Transplantation Humans Lymphocytes Lymphocytes - metabolism Male Medical sciences Middle Aged Mortality Multivariate Analysis Pathogenesis Postoperative Complications - epidemiology Postoperative Complications - etiology Postoperative Complications - metabolism Predictive Value of Tests Receptor, Angiotensin, Type 1 - biosynthesis Receptors, Angiotensin - biosynthesis Risk Factors RNA, Messenger - metabolism Spleen Studies Tissue Donors Transplants & implants Treatment Outcome Tunica Intima - diagnostic imaging Tunica Intima - metabolism Tunica Intima - pathology Ultrasonography, Interventional |
| title | Angiotensin II receptors from peritransplantation through first-year post-transplantation and the risk of transplant coronary artery disease |
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