Isolation and characterization of human esophageal microvascular endothelial cells: mechanisms of inflammatory activation

Gastroesophageal reflux disease is the most common malady of the esophagus, affecting 7% of the United States population. Histological assessment demonstrates classic inflammatory mechanisms including selective leukocyte recruitment and hemorrhage, suggesting a prominent role for the microvasculatur...

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Veröffentlicht in:	American journal of physiology: Gastrointestinal and liver physiology 2003-12, Vol.285 (6), p.G1277-G1292
Hauptverfasser:	Rafiee, Parvaneh, Ogawa, Hitoshi, Heidemann, Jan, Li, Mona S, Aslam, Mohammed, Lamirand, Thomas H, Fisher, Pamela J, Graewin, Shannon J, Dwinell, Michael B, Johnson, Christopher P, Shaker, Reza, Binion, David G
Format:	Artikel
Sprache:	eng
Schlagworte:	Cell Adhesion Cell Adhesion Molecules - metabolism Cell Separation Cells, Cultured Chemokines - metabolism Curcumin - pharmacology Endothelium, Vascular - cytology Endothelium, Vascular - metabolism Endothelium, Vascular - physiology Enzyme Activation - drug effects Enzyme Inhibitors - pharmacology Esophagitis - etiology Esophagus - blood supply Humans Immunoglobulins - metabolism Intestines - blood supply JNK Mitogen-Activated Protein Kinases Leukocytes - physiology Microcirculation Mitogen-Activated Protein Kinases - metabolism Mucoproteins - metabolism NF-kappa B - metabolism Nitric Oxide - biosynthesis Nitric Oxide Synthase - metabolism Nitric Oxide Synthase Type II Phenotype Signal Transduction Vascular Cell Adhesion Molecule-1 - drug effects
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