Distinct inflammatory mechanisms mediate early versus late colitis in mice

Progression from the acute to chronic phase of inflammatory bowel disease cannot be easily evaluated in patients and has not been characterized in animal models. We report a longitudinal study investigating changes in the mucosal immune response in an experimental model of colitis. Severity of colit...

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Veröffentlicht in:	Gastroenterology (New York, N.Y. 1943) N.Y. 1943), 2002-01, Vol.122 (1), p.94-105
Hauptverfasser:	Spencer, David M., Veldman, Geertruidam M., Banerjee, Subhashis, Willis, Joseph, Levine, Alan D.
Format:	Artikel
Sprache:	eng
Schlagworte:	Acute Disease Animals Antibodies - pharmacology Apolipoproteins - metabolism Biological and medical sciences Cells, Cultured Chronic Disease Colitis - immunology Colitis - metabolism Diarrhea - immunology Diarrhea - metabolism Disease Models, Animal Gastric Mucosa - immunology Gastric Mucosa - metabolism Gastroenterology. Liver. Pancreas. Abdomen Interferon-gamma - biosynthesis Interferon-gamma - immunology Interleukin-1 - biosynthesis Interleukin-10 - genetics Interleukin-13 - biosynthesis Interleukin-18 - genetics Interleukin-18 - metabolism Interleukin-4 - biosynthesis Leukocytes - cytology Leukocytes - immunology Leukocytes - metabolism Medical sciences Mice Mice, Inbred C57BL Mice, Knockout Occult Blood Other diseases. Semiology Receptors, Interleukin - biosynthesis Receptors, Interleukin - immunology Receptors, Interleukin-12 RNA, Messenger - analysis Serum Amyloid A Protein - metabolism Stomach. Duodenum. Small intestine. Colon. Rectum. Anus Tumor Necrosis Factor-alpha - biosynthesis
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