Tumor necrosis factor receptor p55 and p75 deficiency protects mice from developing experimental autoimmune myasthenia gravis

The precise pathogenic role of proinflammatory cytokines belonging to the tumor necrosis factor (TNF) family has not been investigated yet in antibody-mediated myasthenia gravis (MG) and experimental autoimmune myasthenia gravis (EAMG). In this study we report that TNF receptor p55 −/− p75 −/− mice...

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Veröffentlicht in:	Journal of neuroimmunology 2002, Vol.122 (1), p.85-93
Hauptverfasser:	Goluszko, Elzbieta, Deng, Caishu, Poussin, Mathilde A, Christadoss, Premkumar
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Antigens, CD - genetics Autoimmunity experimental myasthenia gravis Female Genetic Predisposition to Disease Immunization Immunoglobulin Class Switching - immunology Immunoglobulin G - immunology Immunoglobulin M - immunology Lymphocytes - immunology Lymphocytes - metabolism Lymphotoxin-alpha - immunology Lymphotoxin-α Male Mice Mice, Inbred C57BL Mice, Mutant Strains Muscle, Skeletal - chemistry Muscle, Skeletal - immunology Myasthenia gravis Myasthenia Gravis, Autoimmune, Experimental - genetics Myasthenia Gravis, Autoimmune, Experimental - immunology p55 protein p75 protein Receptors, Cholinergic - chemistry Receptors, Cholinergic - immunology Receptors, Tumor Necrosis Factor - genetics Receptors, Tumor Necrosis Factor, Type I Receptors, Tumor Necrosis Factor, Type II TNF receptors TNF-α Tumor Necrosis Factor-alpha - metabolism
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