Crow-Fukase Syndrome with Ischemic Cardiomyopathy
A 31-year-old man was admitted to our hospital for further evaluation of heart failure symptoms. Crow-Fukase syndrome was diagnosed on the basis of findings of polyneuropathy, hepatomegaly, monoclonal hypergammaglobulinemia, and hypertrichosis. Dipyridamole-stress thallium-201 perfusion imaging, con...
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Veröffentlicht in: | Internal Medicine 2001, Vol.40(8), pp.726-730 |
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creator | ICHIKAWA, Yoshihiko NAKATA, Tomoaki OHHATA, Junichi WAKABAYASHI, Takeru SASAO, Hisataka TSUCHIHASHI, Kazufumi SHIMAMOTO, Kazuaki CHIBA, Susumu MATSUMOTO, Hiroyuki |
description | A 31-year-old man was admitted to our hospital for further evaluation of heart failure symptoms. Crow-Fukase syndrome was diagnosed on the basis of findings of polyneuropathy, hepatomegaly, monoclonal hypergammaglobulinemia, and hypertrichosis. Dipyridamole-stress thallium-201 perfusion imaging, contrast left ventriculography, and coronary angiography revealed a markedly dilated and dysfunctioning left ventricle, extensive reversible ischemia with fixed defect, and multiple coronary lesions. Histopathology of myocardial biopsy specimens demonstrated ischemia-induced myocardial necrosis. These findings suggested that ischemic cardiomyopathy, probably due to inflammatory reactions of coronary arteries in Crow-Fukase syndrome, was responsible for the heart failure symptoms and left ventricular dysfunction in this patient. (Internal Medicine 40: 726-730, 2001) |
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Crow-Fukase syndrome was diagnosed on the basis of findings of polyneuropathy, hepatomegaly, monoclonal hypergammaglobulinemia, and hypertrichosis. Dipyridamole-stress thallium-201 perfusion imaging, contrast left ventriculography, and coronary angiography revealed a markedly dilated and dysfunctioning left ventricle, extensive reversible ischemia with fixed defect, and multiple coronary lesions. Histopathology of myocardial biopsy specimens demonstrated ischemia-induced myocardial necrosis. These findings suggested that ischemic cardiomyopathy, probably due to inflammatory reactions of coronary arteries in Crow-Fukase syndrome, was responsible for the heart failure symptoms and left ventricular dysfunction in this patient. (Internal Medicine 40: 726-730, 2001)</description><identifier>ISSN: 0918-2918</identifier><identifier>EISSN: 1349-7235</identifier><identifier>DOI: 10.2169/internalmedicine.40.726</identifier><identifier>PMID: 11518111</identifier><language>eng</language><publisher>Tokyo: The Japanese Society of Internal Medicine</publisher><subject>Adult ; Biological and medical sciences ; Cardiovascular system ; congestive heart failure ; Coronary Angiography ; coronary artery disease ; Dipyridamole ; Electrocardiography ; Humans ; Investigative techniques, diagnostic techniques (general aspects) ; Male ; Medical sciences ; myocardial ischemia ; Myocardial Ischemia - complications ; Myocardial Ischemia - diagnosis ; Myocardial Ischemia - etiology ; Myocardial Ischemia - physiopathology ; POEMS syndrome ; POEMS Syndrome - complications ; POEMS Syndrome - diagnosis ; POEMS Syndrome - diagnostic imaging ; POEMS Syndrome - physiopathology ; Radiodiagnosis. Nmr imagery. Nmr spectrometry ; Radionuclide Imaging ; Thallium ; Vasodilator Agents ; Ventricular Dysfunction, Left - diagnosis ; Ventricular Dysfunction, Left - etiology ; Ventricular Dysfunction, Left - physiopathology</subject><ispartof>Internal Medicine, 2001, Vol.40(8), pp.726-730</ispartof><rights>The Japanese Society of Internal Medicine</rights><rights>2002 INIST-CNRS</rights><lds50>peer_reviewed</lds50><oa>free_for_read</oa><woscitedreferencessubscribed>false</woscitedreferencessubscribed><citedby>FETCH-LOGICAL-c502t-b4daf80aaad4bb6460aa21c7d1144f73adf65992b12ca2e78709dd8038027e743</citedby></display><links><openurl>$$Topenurl_article</openurl><openurlfulltext>$$Topenurlfull_article</openurlfulltext><thumbnail>$$Tsyndetics_thumb_exl</thumbnail><link.rule.ids>314,780,784,1881,27915,27916</link.rule.ids><backlink>$$Uhttp://pascal-francis.inist.fr/vibad/index.php?action=getRecordDetail&idt=14072050$$DView record in Pascal Francis$$Hfree_for_read</backlink><backlink>$$Uhttps://www.ncbi.nlm.nih.gov/pubmed/11518111$$D View this record in MEDLINE/PubMed$$Hfree_for_read</backlink></links><search><creatorcontrib>ICHIKAWA, Yoshihiko</creatorcontrib><creatorcontrib>NAKATA, Tomoaki</creatorcontrib><creatorcontrib>OHHATA, Junichi</creatorcontrib><creatorcontrib>WAKABAYASHI, Takeru</creatorcontrib><creatorcontrib>SASAO, Hisataka</creatorcontrib><creatorcontrib>TSUCHIHASHI, Kazufumi</creatorcontrib><creatorcontrib>SHIMAMOTO, Kazuaki</creatorcontrib><creatorcontrib>CHIBA, Susumu</creatorcontrib><creatorcontrib>MATSUMOTO, Hiroyuki</creatorcontrib><title>Crow-Fukase Syndrome with Ischemic Cardiomyopathy</title><title>Internal Medicine</title><addtitle>Intern. Med.</addtitle><description>A 31-year-old man was admitted to our hospital for further evaluation of heart failure symptoms. Crow-Fukase syndrome was diagnosed on the basis of findings of polyneuropathy, hepatomegaly, monoclonal hypergammaglobulinemia, and hypertrichosis. Dipyridamole-stress thallium-201 perfusion imaging, contrast left ventriculography, and coronary angiography revealed a markedly dilated and dysfunctioning left ventricle, extensive reversible ischemia with fixed defect, and multiple coronary lesions. Histopathology of myocardial biopsy specimens demonstrated ischemia-induced myocardial necrosis. These findings suggested that ischemic cardiomyopathy, probably due to inflammatory reactions of coronary arteries in Crow-Fukase syndrome, was responsible for the heart failure symptoms and left ventricular dysfunction in this patient. (Internal Medicine 40: 726-730, 2001)</description><subject>Adult</subject><subject>Biological and medical sciences</subject><subject>Cardiovascular system</subject><subject>congestive heart failure</subject><subject>Coronary Angiography</subject><subject>coronary artery disease</subject><subject>Dipyridamole</subject><subject>Electrocardiography</subject><subject>Humans</subject><subject>Investigative techniques, diagnostic techniques (general aspects)</subject><subject>Male</subject><subject>Medical sciences</subject><subject>myocardial ischemia</subject><subject>Myocardial Ischemia - complications</subject><subject>Myocardial Ischemia - diagnosis</subject><subject>Myocardial Ischemia - etiology</subject><subject>Myocardial Ischemia - physiopathology</subject><subject>POEMS syndrome</subject><subject>POEMS Syndrome - complications</subject><subject>POEMS Syndrome - diagnosis</subject><subject>POEMS Syndrome - diagnostic imaging</subject><subject>POEMS Syndrome - physiopathology</subject><subject>Radiodiagnosis. Nmr imagery. Nmr spectrometry</subject><subject>Radionuclide Imaging</subject><subject>Thallium</subject><subject>Vasodilator Agents</subject><subject>Ventricular Dysfunction, Left - diagnosis</subject><subject>Ventricular Dysfunction, Left - etiology</subject><subject>Ventricular Dysfunction, Left - physiopathology</subject><issn>0918-2918</issn><issn>1349-7235</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>2001</creationdate><recordtype>article</recordtype><sourceid>EIF</sourceid><recordid>eNplkE1LAzEQhoMotlb_gvait62ZbHazOUrxCwoK6jnMJlkb3Y-a7FL6701pUdDLTCDPJM-8hFwAnTHI5bVre-tbrBtrnHatnXE6Eyw_IGNIuUwES7NDMqYSioTFMiInIXxQmhZCsmMyAsigAIAxgbnv1snd8InBTl82rfFdY6dr1y-nj0EvbeP0dI7euK7ZdCvsl5tTclRhHezZvk_I293t6_whWTzdP85vFonOKOuTkhusCoqIhpdlzvN4ZKCFAeC8EimaKs-kZCUwjcyKQlBpTBEVKRNW8HRCrnbvrnz3NdjQq8YFbesaW9sNQYnoz6UsIih2oPZdCN5WauVdg36jgKptWupvWopTFdOKk-f7L4Yy3v3O7eOJwOUewKCxrjy22oVfjlPBaEYj97zjPkKP7_YHQN87Xdt_AhAX30oUuxJdflC9RK9sm34DByqVAQ</recordid><startdate>20010801</startdate><enddate>20010801</enddate><creator>ICHIKAWA, Yoshihiko</creator><creator>NAKATA, Tomoaki</creator><creator>OHHATA, Junichi</creator><creator>WAKABAYASHI, Takeru</creator><creator>SASAO, Hisataka</creator><creator>TSUCHIHASHI, Kazufumi</creator><creator>SHIMAMOTO, Kazuaki</creator><creator>CHIBA, Susumu</creator><creator>MATSUMOTO, Hiroyuki</creator><general>The Japanese Society of Internal Medicine</general><general>Japanese Society of Internal Medicine</general><scope>IQODW</scope><scope>CGR</scope><scope>CUY</scope><scope>CVF</scope><scope>ECM</scope><scope>EIF</scope><scope>NPM</scope><scope>AAYXX</scope><scope>CITATION</scope><scope>7X8</scope></search><sort><creationdate>20010801</creationdate><title>Crow-Fukase Syndrome with Ischemic Cardiomyopathy</title><author>ICHIKAWA, Yoshihiko ; NAKATA, Tomoaki ; OHHATA, Junichi ; WAKABAYASHI, Takeru ; SASAO, Hisataka ; TSUCHIHASHI, Kazufumi ; SHIMAMOTO, Kazuaki ; CHIBA, Susumu ; MATSUMOTO, Hiroyuki</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-c502t-b4daf80aaad4bb6460aa21c7d1144f73adf65992b12ca2e78709dd8038027e743</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng</language><creationdate>2001</creationdate><topic>Adult</topic><topic>Biological and medical sciences</topic><topic>Cardiovascular system</topic><topic>congestive heart failure</topic><topic>Coronary Angiography</topic><topic>coronary artery disease</topic><topic>Dipyridamole</topic><topic>Electrocardiography</topic><topic>Humans</topic><topic>Investigative techniques, diagnostic techniques (general aspects)</topic><topic>Male</topic><topic>Medical sciences</topic><topic>myocardial ischemia</topic><topic>Myocardial Ischemia - complications</topic><topic>Myocardial Ischemia - diagnosis</topic><topic>Myocardial Ischemia - etiology</topic><topic>Myocardial Ischemia - physiopathology</topic><topic>POEMS syndrome</topic><topic>POEMS Syndrome - complications</topic><topic>POEMS Syndrome - diagnosis</topic><topic>POEMS Syndrome - diagnostic imaging</topic><topic>POEMS Syndrome - physiopathology</topic><topic>Radiodiagnosis. Nmr imagery. Nmr spectrometry</topic><topic>Radionuclide Imaging</topic><topic>Thallium</topic><topic>Vasodilator Agents</topic><topic>Ventricular Dysfunction, Left - diagnosis</topic><topic>Ventricular Dysfunction, Left - etiology</topic><topic>Ventricular Dysfunction, Left - physiopathology</topic><toplevel>peer_reviewed</toplevel><toplevel>online_resources</toplevel><creatorcontrib>ICHIKAWA, Yoshihiko</creatorcontrib><creatorcontrib>NAKATA, Tomoaki</creatorcontrib><creatorcontrib>OHHATA, Junichi</creatorcontrib><creatorcontrib>WAKABAYASHI, Takeru</creatorcontrib><creatorcontrib>SASAO, Hisataka</creatorcontrib><creatorcontrib>TSUCHIHASHI, Kazufumi</creatorcontrib><creatorcontrib>SHIMAMOTO, Kazuaki</creatorcontrib><creatorcontrib>CHIBA, Susumu</creatorcontrib><creatorcontrib>MATSUMOTO, Hiroyuki</creatorcontrib><collection>Pascal-Francis</collection><collection>Medline</collection><collection>MEDLINE</collection><collection>MEDLINE (Ovid)</collection><collection>MEDLINE</collection><collection>MEDLINE</collection><collection>PubMed</collection><collection>CrossRef</collection><collection>MEDLINE - Academic</collection><jtitle>Internal Medicine</jtitle></facets><delivery><delcategory>Remote Search Resource</delcategory><fulltext>fulltext</fulltext></delivery><addata><au>ICHIKAWA, Yoshihiko</au><au>NAKATA, Tomoaki</au><au>OHHATA, Junichi</au><au>WAKABAYASHI, Takeru</au><au>SASAO, Hisataka</au><au>TSUCHIHASHI, Kazufumi</au><au>SHIMAMOTO, Kazuaki</au><au>CHIBA, Susumu</au><au>MATSUMOTO, Hiroyuki</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>Crow-Fukase Syndrome with Ischemic Cardiomyopathy</atitle><jtitle>Internal Medicine</jtitle><addtitle>Intern. Med.</addtitle><date>2001-08-01</date><risdate>2001</risdate><volume>40</volume><issue>8</issue><spage>726</spage><epage>730</epage><pages>726-730</pages><issn>0918-2918</issn><eissn>1349-7235</eissn><abstract>A 31-year-old man was admitted to our hospital for further evaluation of heart failure symptoms. Crow-Fukase syndrome was diagnosed on the basis of findings of polyneuropathy, hepatomegaly, monoclonal hypergammaglobulinemia, and hypertrichosis. Dipyridamole-stress thallium-201 perfusion imaging, contrast left ventriculography, and coronary angiography revealed a markedly dilated and dysfunctioning left ventricle, extensive reversible ischemia with fixed defect, and multiple coronary lesions. Histopathology of myocardial biopsy specimens demonstrated ischemia-induced myocardial necrosis. These findings suggested that ischemic cardiomyopathy, probably due to inflammatory reactions of coronary arteries in Crow-Fukase syndrome, was responsible for the heart failure symptoms and left ventricular dysfunction in this patient. (Internal Medicine 40: 726-730, 2001)</abstract><cop>Tokyo</cop><pub>The Japanese Society of Internal Medicine</pub><pmid>11518111</pmid><doi>10.2169/internalmedicine.40.726</doi><tpages>5</tpages><oa>free_for_read</oa></addata></record> |
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subjects | Adult Biological and medical sciences Cardiovascular system congestive heart failure Coronary Angiography coronary artery disease Dipyridamole Electrocardiography Humans Investigative techniques, diagnostic techniques (general aspects) Male Medical sciences myocardial ischemia Myocardial Ischemia - complications Myocardial Ischemia - diagnosis Myocardial Ischemia - etiology Myocardial Ischemia - physiopathology POEMS syndrome POEMS Syndrome - complications POEMS Syndrome - diagnosis POEMS Syndrome - diagnostic imaging POEMS Syndrome - physiopathology Radiodiagnosis. Nmr imagery. Nmr spectrometry Radionuclide Imaging Thallium Vasodilator Agents Ventricular Dysfunction, Left - diagnosis Ventricular Dysfunction, Left - etiology Ventricular Dysfunction, Left - physiopathology |
title | Crow-Fukase Syndrome with Ischemic Cardiomyopathy |
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