Nicotine Induces Endothelial TNF-α Expression, Which Mediates Growth Retardation in Vitro

Purpose. Atherosclerosis is understood as the common pathologic manifestation of arterial injury caused by a variety of etiologies. One well-established etiologic agent is nicotine. We hypothesized that cytokines of endothelial origin are involved with the pathologic changes found in atherosclerosis...

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Veröffentlicht in:	The Journal of surgical research 2001-08, Vol.99 (2), p.381-384
Hauptverfasser:	Albaugh, Gregory, Kann, Brian, Strande, Louise, Vemulapalli, Pratibha, Hewitt, Charles, Alexander, James B.
Format:	Artikel
Sprache:	eng
Schlagworte:	Arteriosclerosis - etiology Arteriosclerosis - metabolism atherogenesis Atherosclerosis (general aspects, experimental research) Biological and medical sciences Blood and lymphatic vessels Cardiology. Vascular system Cell Division - drug effects Cells, Cultured cytokines Dose-Response Relationship, Drug endothelium Endothelium, Vascular - cytology Endothelium, Vascular - drug effects Endothelium, Vascular - metabolism Humans Immunohistochemistry In Vitro Techniques Medical sciences nicotine Nicotine - pharmacology Nicotinic Agonists - pharmacology Smoking - adverse effects Tumor Necrosis Factor-alpha - biosynthesis Umbilical Veins - cytology
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