Quantitative immunochemistry on neuronal loss, reactive gliosis and BBB damage in cortex/striatum and hippocampus/amygdala after systemic kainic acid administration

Cell specific markers were quantified in the hippocampus, the amygdala/pyriform cortex, the frontal cerebral cortex and the striatum of the rat brain after systemic administration of kainic acid. Neuron specific enolase (NSE) reflects loss of neurons, glial fibrillary acidic protein (GFAP) reflects...

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Veröffentlicht in:	Neurochemistry international 2000-04, Vol.36 (4), p.313-318
Hauptverfasser:	Ding, Mei, Haglid, Kenneth G, Hamberger, Anders
Format:	Artikel
Sprache:	eng
Schlagworte:	Amygdala - metabolism Animals Blood Proteins - metabolism Blood-Brain Barrier - drug effects Blood–brain barrier Brain - pathology Brain - physiopathology Cell Death - physiology Cerebral Cortex - metabolism Corpus Striatum - metabolism Excitatory Amino Acid Agonists - pharmacology GFAP Glial Fibrillary Acidic Protein - metabolism Gliosis Gliosis - chemically induced Gliosis - metabolism Gliosis - pathology Gliosis - physiopathology Hippocampus - metabolism Immunochemistry Kainic acid Kainic Acid - pharmacology Male Neurodegeneration neuron-specific enolase Neurons - physiology NSE Phosphopyruvate Hydratase - metabolism Rats Rats, Sprague-Dawley S-100 S100 Proteins - metabolism
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