Oxidative stress occurs in absence of hyperglycaemia and inflammation in the onset of kidney lesions in normotensive obese rats

Background. Several factors favour the development of kidney lesions. We examined the role of oxidative stress in the onset of renal alterations that occur in Zucker obese (ZO) fa/fa rats. Methods. Kidney structure, biological data, glycation parameters, advanced glycation end products (AGE), thioba...

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Veröffentlicht in:	Nephrology, dialysis, transplantation dialysis, transplantation, 2000-04, Vol.15 (4), p.467-476
Hauptverfasser:	Poirier, Bruno, Lannaud-Bournoville, Marilyne, Conti, Marc, Bazin, Raymond, Michel, Odile, Bariéty, Jean, Chevalier, Jacques, Myara, Isaac
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Biological and medical sciences Blood Glucose - metabolism Blood Pressure - physiology Cholesterol - blood Disease Models, Animal Fibrosis glomerulosclerosis Glomerulosclerosis, Focal Segmental - metabolism Glomerulosclerosis, Focal Segmental - pathology Glomerulosclerosis, Focal Segmental - physiopathology Glycated Hemoglobin A - metabolism Glycation End Products, Advanced - metabolism Glycosylation Hyperglycemia - blood hyperinsulinaemia Inflammation Kidney Tubules - metabolism Kidney Tubules - pathology lipid peroxidation Lipid Peroxidation - physiology Male Medical sciences Metabolic diseases Obesity Oxidative Stress - physiology Rats Rats, Zucker Thiobarbituric Acid Reactive Substances - metabolism Triglycerides - blood Urodynamics Zucker rat
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