Kupffer Cell Sensitization by Alcohol Involves Increased Permeability to Gut-Derived Endotoxin

Background: Studies with gut sterilization and Kupffer cell inactivation support the hypothesis that endotoxin and Kupffer cells are involved in mechanisms of alcohol‐induced liver injury. Recently, we found that Kupffer cells isolated from rats treated only once with ethanol were sensitized to endo...

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Veröffentlicht in:	Alcoholism, clinical and experimental research clinical and experimental research, 2001-06, Vol.25 (s2), p.51S-54S
Hauptverfasser:	Enomoto, Nobuyuki, Ikejima, Kenichi, Yamashina, Shunhei, Hirose, Miyoko, Shimizu, Hidetake, Kitamura, Tsuneo, Takei, Yoshiyuki, Sato, Nobuhiro, Thurman, Ronald G.
Format:	Artikel
Sprache:	eng
Schlagworte:	Alcoholism and acute alcohol poisoning Animals Anti-Bacterial Agents - pharmacology Antibiotics Biological and medical sciences Calcium - metabolism Digestive System - metabolism Endotoxin Endotoxins - blood Endotoxins - metabolism Endotoxins - pharmacology Ethanol - blood Ethanol - pharmacokinetics Ethanol - pharmacology Female Gut Permeability Horseradish Peroxidase - pharmacokinetics Kupffer Cell Kupffer Cells - drug effects Medical sciences Permeability Portal Vein Rats Rats, Wistar Toxicology Tumor Necrosis Factor-alpha - metabolism
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