Phosphorylation of the flagellar regulatory protein FlrC is necessary for Vibrio cholerae motility and enhanced colonization

The human pathogen Vibrio cholerae specifically expresses virulence factors within the host, including cholera toxin (CT) and the toxin co‐regulated pilus (TCP), which allow it to colonize the intestine and cause disease. V. cholerae is a highly motile organism by virtue of a polar flagellum, and mo...

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Veröffentlicht in:	Molecular microbiology 2000-02, Vol.35 (4), p.743-755
Hauptverfasser:	Correa, Nidia E., Lauriano, Crystal M., McGee, Raynia, Klose, Karl E.
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Aspartic Acid - metabolism Bacterial Adhesion - genetics Bacterial Proteins - genetics Bacterial Proteins - metabolism Cell Division DNA-Binding Proteins DNA-Directed RNA Polymerases - physiology Flagella - metabolism Flagellin - genetics FlrC protein Gene Expression Regulation, Bacterial Hemolysin Proteins - metabolism Humans Mice Movement Mutation Phosphates - metabolism Phosphorylation Phosphotransferases - genetics Phosphotransferases - metabolism RNA Polymerase Sigma 54 Sigma Factor - physiology Transcription, Genetic Vibrio cholerae Vibrio cholerae - genetics Vibrio cholerae - growth & development Vibrio cholerae - metabolism
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