Physiologic transfusion triggers
In clinical practice, the decision to transfuse is linked to the hope of increasing oxygen transport (TO2 ) to tissues. Physiologic transfusion triggers should progressively replace arbitrary hemoglobin-based transfusion triggers. These ‘physiologic’ transfusion triggers can be based on signs and sy...
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| Veröffentlicht in: | Best practice & research. Clinical anaesthesiology 2007-06, Vol.21 (2), p.173-181 |
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| container_title | Best practice & research. Clinical anaesthesiology |
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| creator | Vallet, Benoit, MD, PhD Adamczyk, Sébastien Barreau, Olivier Lebuffe, Gilles, MD, PhD |
| description | In clinical practice, the decision to transfuse is linked to the hope of increasing oxygen transport (TO2 ) to tissues. Physiologic transfusion triggers should progressively replace arbitrary hemoglobin-based transfusion triggers. These ‘physiologic’ transfusion triggers can be based on signs and symptoms of impaired global oxygenation (lactate, venous O2 saturation [SvO2 ]) or, even better, of regional tissue oxygenation (electrocardiographic ST-segment, electroencephalographic P300 latency). The SvO2 or its surrogate, the central venous O2 saturation (ScvO2 ), is a clinical tool which integrates the relationship between whole-body O2 uptake and TO2 , and as such can be proposed as a simple physiologic transfusion trigger. |
| doi_str_mv | 10.1016/j.bpa.2007.02.003 |
| format | Article |
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Clinical anaesthesiology</title><addtitle>Best Pract Res Clin Anaesthesiol</addtitle><description>In clinical practice, the decision to transfuse is linked to the hope of increasing oxygen transport (TO2 ) to tissues. Physiologic transfusion triggers should progressively replace arbitrary hemoglobin-based transfusion triggers. These ‘physiologic’ transfusion triggers can be based on signs and symptoms of impaired global oxygenation (lactate, venous O2 saturation [SvO2 ]) or, even better, of regional tissue oxygenation (electrocardiographic ST-segment, electroencephalographic P300 latency). The SvO2 or its surrogate, the central venous O2 saturation (ScvO2 ), is a clinical tool which integrates the relationship between whole-body O2 uptake and TO2 , and as such can be proposed as a simple physiologic transfusion trigger.</description><subject>Anesthesia & Perioperative Care</subject><subject>Blood Transfusion</subject><subject>Catheterization, Central Venous</subject><subject>central venous O 2 saturation (ScvO 2)</subject><subject>Clinical Protocols</subject><subject>Critical Illness - classification</subject><subject>Critical Illness - therapy</subject><subject>Decision Making</subject><subject>electroencephalographic P300 latency</subject><subject>Hemoglobins - metabolism</subject><subject>Humans</subject><subject>lactate</subject><subject>Monitoring, Physiologic - methods</subject><subject>Oximetry</subject><subject>oxygen (O 2) transport (TO 2)</subject><subject>Oxygen - blood</subject><subject>Oxygen Consumption</subject><subject>Reference Values</subject><subject>regional tissue oxygenation</subject><subject>Resuscitation</subject><subject>Transfusion Reaction</subject><subject>Veins</subject><subject>venous O 2 saturation (SvO 2)</subject><issn>1521-6896</issn><issn>1532-169X</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>2007</creationdate><recordtype>article</recordtype><sourceid>EIF</sourceid><recordid>eNp9kU9LxDAQxYMorq5-AC-yJ2-tk6RNGgRBFv_BgoIK3kKapGtqt12TVthvb8ouCB48zQy892B-D6EzDCkGzC7rtFyrlADwFEgKQPfQEc4pSTAT7_vjTnDCCsEm6DiEGgAwL8QhmmDOcuAcjtDs-WMTXNd0S6dnvVdtqIZ4t3F3y6X14QQdVKoJ9nQ3p-jt7vZ1_pAsnu4f5zeLRGfA-kRohS0xOMaW2ghigEFmTM40I7nABcVZRlVeclpQbbKCgxGaCF4pZoQBTqfoYpu79t3XYEMvVy5o2zSqtd0QJAeesZgShXgr1L4LwdtKrr1bKb-RGOSIRdYyYpEjFglERizRc74LH8qVNb-OHYcouNoKbHzx21kvg3a21dY4b3UvTef-jb_-49aNa51Wzafd2FB3g28jO4lliAb5MvYy1gJ8rETk9Ae-MoXd</recordid><startdate>20070601</startdate><enddate>20070601</enddate><creator>Vallet, Benoit, MD, PhD</creator><creator>Adamczyk, Sébastien</creator><creator>Barreau, Olivier</creator><creator>Lebuffe, Gilles, MD, PhD</creator><general>Elsevier Ltd</general><scope>CGR</scope><scope>CUY</scope><scope>CVF</scope><scope>ECM</scope><scope>EIF</scope><scope>NPM</scope><scope>AAYXX</scope><scope>CITATION</scope><scope>7X8</scope></search><sort><creationdate>20070601</creationdate><title>Physiologic transfusion triggers</title><author>Vallet, Benoit, MD, PhD ; Adamczyk, Sébastien ; Barreau, Olivier ; Lebuffe, Gilles, MD, PhD</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-c406t-9ca1e2d1507bcd92d0604dd56c62591831443a5b7383cd4870d9c297fa6d9d073</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng</language><creationdate>2007</creationdate><topic>Anesthesia & Perioperative Care</topic><topic>Blood Transfusion</topic><topic>Catheterization, Central Venous</topic><topic>central venous O 2 saturation (ScvO 2)</topic><topic>Clinical Protocols</topic><topic>Critical Illness - classification</topic><topic>Critical Illness - therapy</topic><topic>Decision Making</topic><topic>electroencephalographic P300 latency</topic><topic>Hemoglobins - metabolism</topic><topic>Humans</topic><topic>lactate</topic><topic>Monitoring, Physiologic - methods</topic><topic>Oximetry</topic><topic>oxygen (O 2) transport (TO 2)</topic><topic>Oxygen - blood</topic><topic>Oxygen Consumption</topic><topic>Reference Values</topic><topic>regional tissue oxygenation</topic><topic>Resuscitation</topic><topic>Transfusion Reaction</topic><topic>Veins</topic><topic>venous O 2 saturation (SvO 2)</topic><toplevel>peer_reviewed</toplevel><toplevel>online_resources</toplevel><creatorcontrib>Vallet, Benoit, MD, PhD</creatorcontrib><creatorcontrib>Adamczyk, Sébastien</creatorcontrib><creatorcontrib>Barreau, Olivier</creatorcontrib><creatorcontrib>Lebuffe, Gilles, MD, PhD</creatorcontrib><collection>Medline</collection><collection>MEDLINE</collection><collection>MEDLINE (Ovid)</collection><collection>MEDLINE</collection><collection>MEDLINE</collection><collection>PubMed</collection><collection>CrossRef</collection><collection>MEDLINE - Academic</collection><jtitle>Best practice & research. 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These ‘physiologic’ transfusion triggers can be based on signs and symptoms of impaired global oxygenation (lactate, venous O2 saturation [SvO2 ]) or, even better, of regional tissue oxygenation (electrocardiographic ST-segment, electroencephalographic P300 latency). The SvO2 or its surrogate, the central venous O2 saturation (ScvO2 ), is a clinical tool which integrates the relationship between whole-body O2 uptake and TO2 , and as such can be proposed as a simple physiologic transfusion trigger.</abstract><cop>Netherlands</cop><pub>Elsevier Ltd</pub><pmid>17650770</pmid><doi>10.1016/j.bpa.2007.02.003</doi><tpages>9</tpages></addata></record> |
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| ispartof | Best practice & research. Clinical anaesthesiology, 2007-06, Vol.21 (2), p.173-181 |
| issn | 1521-6896 1532-169X |
| language | eng |
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| source | MEDLINE; Elsevier ScienceDirect Journals |
| subjects | Anesthesia & Perioperative Care Blood Transfusion Catheterization, Central Venous central venous O 2 saturation (ScvO 2) Clinical Protocols Critical Illness - classification Critical Illness - therapy Decision Making electroencephalographic P300 latency Hemoglobins - metabolism Humans lactate Monitoring, Physiologic - methods Oximetry oxygen (O 2) transport (TO 2) Oxygen - blood Oxygen Consumption Reference Values regional tissue oxygenation Resuscitation Transfusion Reaction Veins venous O 2 saturation (SvO 2) |
| title | Physiologic transfusion triggers |
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