Mitochondrial genetic background modifies breast cancer risk

Inefficient mitochondrial electron transport chain (ETC) function has been implicated in the vicious cycle of reactive oxygen species (ROS) production that may predispose an individual to late onset diseases, such as diabetes, hypertension, and cancer. Mitochondrial DNA (mtDNA) variations may affect...

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Veröffentlicht in:	Cancer research (Chicago, Ill.) Ill.), 2007-05, Vol.67 (10), p.4687-4694
Hauptverfasser:	BAI, Ren-Kui, LEAL, Suzanne M, COVARRUBIAS, Daniel, AIYI LIU, WONG, Lee-Jun C
Format:	Artikel
Sprache:	eng
Schlagworte:	Adult Aged Aged, 80 and over Antineoplastic agents Biological and medical sciences Breast Neoplasms - genetics Breast Neoplasms - metabolism Case-Control Studies DNA, Mitochondrial - genetics Electron Transport Female Genetic Predisposition to Disease Gynecology. Andrology. Obstetrics Humans Mammary gland diseases Medical sciences Middle Aged Mitochondria - genetics Mitochondria - metabolism Pharmacology. Drug treatments Polymorphism, Single Nucleotide Reactive Oxygen Species - metabolism Tumors
Online-Zugang:	Volltext
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