Nitric Oxide Evokes an Adaptive Response to Oxidative Stress by Arresting Respiration

Aerobic metabolism generates biologically challenging reactive oxygen species (ROS) by the endogenous autooxidation of components of the electron transport chain (ETC). Basal levels of oxidative stress can dramatically rise upon activation of the NADPH oxidase-dependent respiratory burst. To minimiz...

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Veröffentlicht in:	The Journal of biological chemistry 2008-03, Vol.283 (12), p.7682-7689
Hauptverfasser:	Husain, Maroof, Bourret, Travis J., McCollister, Bruce D., Jones-Carson, Jessica, Laughlin, James, Vázquez-Torres, Andrés
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Bacterial Proteins - genetics Bacterial Proteins - metabolism Drug Resistance, Microbial - genetics Electron Transport Chain Complex Proteins - genetics Electron Transport Chain Complex Proteins - metabolism Interferon-gamma - pharmacology Macrophages, Peritoneal - enzymology Macrophages, Peritoneal - microbiology Membrane Glycoproteins - genetics Membrane Glycoproteins - metabolism Mice Mice, Knockout NADPH Dehydrogenase - genetics NADPH Dehydrogenase - metabolism NADPH Oxidase 2 NADPH Oxidases - genetics NADPH Oxidases - metabolism Nitric Oxide - metabolism Nitric Oxide Synthase Type II - genetics Nitric Oxide Synthase Type II - metabolism Oxidative Stress - genetics Peroxiredoxins - genetics Peroxiredoxins - metabolism Peroxynitrous Acid - pharmacology Reactive Oxygen Species - metabolism Respiratory Burst - genetics Salmonella Salmonella Infections, Animal - enzymology Salmonella Infections, Animal - genetics Salmonella typhimurium - enzymology Salmonella typhimurium - genetics
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