Response of the Heart and Pulmonary Arteries to Hypoxia, Pressure, and Volume. A Short Review
The pulmonary arterioles react to hypoxia by contraction and to increased pressure and volume by hypertrophy of the muscular wall, referred to as pulmonary vascular remodeling, both of which increase vascular resistance and result in increased pulmonary arterial pressure. Heart muscle reacts to incr...
Gespeichert in:
| Veröffentlicht in: | Poultry science 2007-05, Vol.86 (5), p.1006-1011 |
|---|---|
| 1. Verfasser: | |
| Format: | Artikel |
| Sprache: | eng |
| Schlagworte: | |
| Online-Zugang: | Volltext |
| Tags: |
Tag hinzufügen
Keine Tags, Fügen Sie den ersten Tag hinzu!
|
| container_end_page | 1011 |
|---|---|
| container_issue | 5 |
| container_start_page | 1006 |
| container_title | Poultry science |
| container_volume | 86 |
| creator | Julian, R.J |
| description | The pulmonary arterioles react to hypoxia by contraction and to increased pressure and volume by hypertrophy of the muscular wall, referred to as pulmonary vascular remodeling, both of which increase vascular resistance and result in increased pulmonary arterial pressure. Heart muscle reacts to increased pressure by hypertrophy of cardiac myocytes and thickening of the muscular wall. The heart responds to increased volume by dilation of the chamber that may result in physiologic or pathologic hypertrophy of the muscle wall. Heart muscle cells are very sensitive to hypoxia or other insults, and this may result in death of individual cardiac myocytes with lengthening and thinning of the remaining heart muscle cells and dilation of the chamber in a process called dilated cardiomyopathy. |
| doi_str_mv | 10.1093/ps/86.5.1006 |
| format | Article |
| fullrecord | <record><control><sourceid>proquest_cross</sourceid><recordid>TN_cdi_proquest_miscellaneous_70385613</recordid><sourceformat>XML</sourceformat><sourcesystem>PC</sourcesystem><sourcerecordid>70385613</sourcerecordid><originalsourceid>FETCH-LOGICAL-c444t-4d8bb1326386ab7ab6045b09c8cc951f2562f36831289b0a82a05ce5473c44403</originalsourceid><addsrcrecordid>eNpdkM1r3DAQR0VoSLab3HJuRQ49rTczkiXLxyW03UCgIR-9FSF7x42DbTmSnTb_fb3ZhUBPPwYej-ExdoawRMjlRR8vjF6q6QB9wGaohEokZviBzQCkSFSW4zH7GOMTgECtsyN2jFkqFch8xn7dUux9F4n7ig-PxNfkwsBdt-E3Y9P6zoVXvgoDhZoiHzxfv_b-b-0W_CZQjGOgxRv80zdjS0u-4nePfhLc0ktNf07YYeWaSKf7nbOHb1_vL9fJ9Y_vV5er66RM03RI0o0pCpRCS6NdkblCQ6oKyEtTlrnCSigtKqmNRGHyApwRDlRJKs3kVgByzr7svH3wzyPFwbZ1LKlpXEd-jDYDaZRGOYHn_4FPfgzd9JsVQqKS6TRztthBZfAxBqpsH-p2CmER7La57aM12iq7bT7hn_bOsWhp8w7vI0_A5x1QOW_d71BH-3AnACVAZhAFyH-XsYMi</addsrcrecordid><sourcetype>Aggregation Database</sourcetype><iscdi>true</iscdi><recordtype>article</recordtype><pqid>223153422</pqid></control><display><type>article</type><title>Response of the Heart and Pulmonary Arteries to Hypoxia, Pressure, and Volume. A Short Review</title><source>MEDLINE</source><source>EZB-FREE-00999 freely available EZB journals</source><source>Alma/SFX Local Collection</source><creator>Julian, R.J</creator><creatorcontrib>Julian, R.J</creatorcontrib><description>The pulmonary arterioles react to hypoxia by contraction and to increased pressure and volume by hypertrophy of the muscular wall, referred to as pulmonary vascular remodeling, both of which increase vascular resistance and result in increased pulmonary arterial pressure. Heart muscle reacts to increased pressure by hypertrophy of cardiac myocytes and thickening of the muscular wall. The heart responds to increased volume by dilation of the chamber that may result in physiologic or pathologic hypertrophy of the muscle wall. Heart muscle cells are very sensitive to hypoxia or other insults, and this may result in death of individual cardiac myocytes with lengthening and thinning of the remaining heart muscle cells and dilation of the chamber in a process called dilated cardiomyopathy.</description><identifier>ISSN: 0032-5791</identifier><identifier>EISSN: 1525-3171</identifier><identifier>DOI: 10.1093/ps/86.5.1006</identifier><identifier>PMID: 17435039</identifier><language>eng</language><publisher>England: Oxford University Press</publisher><subject>Animals ; apoptosis ; blood pressure ; blood vessels ; broiler chickens ; cardiomyopathy ; dilated cardiomyopathy ; heart ; hypertrophy ; Hypertrophy - pathology ; Hypertrophy - physiopathology ; hypoxia ; Hypoxia - metabolism ; literature reviews ; Myocardium - metabolism ; myocytes ; Pressure ; pulmonary arterioles ; pulmonary artery ; Pulmonary Artery - anatomy & histology ; Pulmonary Artery - pathology ; Pulmonary Artery - physiology ; Pulmonary Artery - physiopathology ; vascular resistance ; vasoconstriction ; volume</subject><ispartof>Poultry science, 2007-05, Vol.86 (5), p.1006-1011</ispartof><rights>Copyright Poultry Science Association May 2007</rights><lds50>peer_reviewed</lds50><oa>free_for_read</oa><woscitedreferencessubscribed>false</woscitedreferencessubscribed><citedby>FETCH-LOGICAL-c444t-4d8bb1326386ab7ab6045b09c8cc951f2562f36831289b0a82a05ce5473c44403</citedby><cites>FETCH-LOGICAL-c444t-4d8bb1326386ab7ab6045b09c8cc951f2562f36831289b0a82a05ce5473c44403</cites></display><links><openurl>$$Topenurl_article</openurl><openurlfulltext>$$Topenurlfull_article</openurlfulltext><thumbnail>$$Tsyndetics_thumb_exl</thumbnail><link.rule.ids>314,780,784,27924,27925</link.rule.ids><backlink>$$Uhttps://www.ncbi.nlm.nih.gov/pubmed/17435039$$D View this record in MEDLINE/PubMed$$Hfree_for_read</backlink></links><search><creatorcontrib>Julian, R.J</creatorcontrib><title>Response of the Heart and Pulmonary Arteries to Hypoxia, Pressure, and Volume. A Short Review</title><title>Poultry science</title><addtitle>Poult Sci</addtitle><description>The pulmonary arterioles react to hypoxia by contraction and to increased pressure and volume by hypertrophy of the muscular wall, referred to as pulmonary vascular remodeling, both of which increase vascular resistance and result in increased pulmonary arterial pressure. Heart muscle reacts to increased pressure by hypertrophy of cardiac myocytes and thickening of the muscular wall. The heart responds to increased volume by dilation of the chamber that may result in physiologic or pathologic hypertrophy of the muscle wall. Heart muscle cells are very sensitive to hypoxia or other insults, and this may result in death of individual cardiac myocytes with lengthening and thinning of the remaining heart muscle cells and dilation of the chamber in a process called dilated cardiomyopathy.</description><subject>Animals</subject><subject>apoptosis</subject><subject>blood pressure</subject><subject>blood vessels</subject><subject>broiler chickens</subject><subject>cardiomyopathy</subject><subject>dilated cardiomyopathy</subject><subject>heart</subject><subject>hypertrophy</subject><subject>Hypertrophy - pathology</subject><subject>Hypertrophy - physiopathology</subject><subject>hypoxia</subject><subject>Hypoxia - metabolism</subject><subject>literature reviews</subject><subject>Myocardium - metabolism</subject><subject>myocytes</subject><subject>Pressure</subject><subject>pulmonary arterioles</subject><subject>pulmonary artery</subject><subject>Pulmonary Artery - anatomy & histology</subject><subject>Pulmonary Artery - pathology</subject><subject>Pulmonary Artery - physiology</subject><subject>Pulmonary Artery - physiopathology</subject><subject>vascular resistance</subject><subject>vasoconstriction</subject><subject>volume</subject><issn>0032-5791</issn><issn>1525-3171</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>2007</creationdate><recordtype>article</recordtype><sourceid>EIF</sourceid><sourceid>ABUWG</sourceid><sourceid>AFKRA</sourceid><sourceid>AZQEC</sourceid><sourceid>BENPR</sourceid><sourceid>CCPQU</sourceid><sourceid>DWQXO</sourceid><sourceid>GNUQQ</sourceid><recordid>eNpdkM1r3DAQR0VoSLab3HJuRQ49rTczkiXLxyW03UCgIR-9FSF7x42DbTmSnTb_fb3ZhUBPPwYej-ExdoawRMjlRR8vjF6q6QB9wGaohEokZviBzQCkSFSW4zH7GOMTgECtsyN2jFkqFch8xn7dUux9F4n7ig-PxNfkwsBdt-E3Y9P6zoVXvgoDhZoiHzxfv_b-b-0W_CZQjGOgxRv80zdjS0u-4nePfhLc0ktNf07YYeWaSKf7nbOHb1_vL9fJ9Y_vV5er66RM03RI0o0pCpRCS6NdkblCQ6oKyEtTlrnCSigtKqmNRGHyApwRDlRJKs3kVgByzr7svH3wzyPFwbZ1LKlpXEd-jDYDaZRGOYHn_4FPfgzd9JsVQqKS6TRztthBZfAxBqpsH-p2CmER7La57aM12iq7bT7hn_bOsWhp8w7vI0_A5x1QOW_d71BH-3AnACVAZhAFyH-XsYMi</recordid><startdate>20070501</startdate><enddate>20070501</enddate><creator>Julian, R.J</creator><general>Oxford University Press</general><scope>FBQ</scope><scope>CGR</scope><scope>CUY</scope><scope>CVF</scope><scope>ECM</scope><scope>EIF</scope><scope>NPM</scope><scope>AAYXX</scope><scope>CITATION</scope><scope>3V.</scope><scope>7X2</scope><scope>7X7</scope><scope>7XB</scope><scope>88E</scope><scope>8FE</scope><scope>8FG</scope><scope>8FH</scope><scope>8FI</scope><scope>8FJ</scope><scope>8FK</scope><scope>ABJCF</scope><scope>ABUWG</scope><scope>AFKRA</scope><scope>ATCPS</scope><scope>AZQEC</scope><scope>BENPR</scope><scope>BGLVJ</scope><scope>BHPHI</scope><scope>CCPQU</scope><scope>DWQXO</scope><scope>FYUFA</scope><scope>GHDGH</scope><scope>GNUQQ</scope><scope>HCIFZ</scope><scope>K9.</scope><scope>L6V</scope><scope>M0K</scope><scope>M0S</scope><scope>M1P</scope><scope>M7S</scope><scope>PATMY</scope><scope>PQEST</scope><scope>PQQKQ</scope><scope>PQUKI</scope><scope>PRINS</scope><scope>PTHSS</scope><scope>PYCSY</scope><scope>S0X</scope><scope>7X8</scope></search><sort><creationdate>20070501</creationdate><title>Response of the Heart and Pulmonary Arteries to Hypoxia, Pressure, and Volume. A Short Review</title><author>Julian, R.J</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-c444t-4d8bb1326386ab7ab6045b09c8cc951f2562f36831289b0a82a05ce5473c44403</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng</language><creationdate>2007</creationdate><topic>Animals</topic><topic>apoptosis</topic><topic>blood pressure</topic><topic>blood vessels</topic><topic>broiler chickens</topic><topic>cardiomyopathy</topic><topic>dilated cardiomyopathy</topic><topic>heart</topic><topic>hypertrophy</topic><topic>Hypertrophy - pathology</topic><topic>Hypertrophy - physiopathology</topic><topic>hypoxia</topic><topic>Hypoxia - metabolism</topic><topic>literature reviews</topic><topic>Myocardium - metabolism</topic><topic>myocytes</topic><topic>Pressure</topic><topic>pulmonary arterioles</topic><topic>pulmonary artery</topic><topic>Pulmonary Artery - anatomy & histology</topic><topic>Pulmonary Artery - pathology</topic><topic>Pulmonary Artery - physiology</topic><topic>Pulmonary Artery - physiopathology</topic><topic>vascular resistance</topic><topic>vasoconstriction</topic><topic>volume</topic><toplevel>peer_reviewed</toplevel><toplevel>online_resources</toplevel><creatorcontrib>Julian, R.J</creatorcontrib><collection>AGRIS</collection><collection>Medline</collection><collection>MEDLINE</collection><collection>MEDLINE (Ovid)</collection><collection>MEDLINE</collection><collection>MEDLINE</collection><collection>PubMed</collection><collection>CrossRef</collection><collection>ProQuest Central (Corporate)</collection><collection>Agricultural Science Collection</collection><collection>Health & Medical Collection</collection><collection>ProQuest Central (purchase pre-March 2016)</collection><collection>Medical Database (Alumni Edition)</collection><collection>ProQuest SciTech Collection</collection><collection>ProQuest Technology Collection</collection><collection>ProQuest Natural Science Collection</collection><collection>Hospital Premium Collection</collection><collection>Hospital Premium Collection (Alumni Edition)</collection><collection>ProQuest Central (Alumni) (purchase pre-March 2016)</collection><collection>Materials Science & Engineering Collection</collection><collection>ProQuest Central (Alumni Edition)</collection><collection>ProQuest Central UK/Ireland</collection><collection>Agricultural & Environmental Science Collection</collection><collection>ProQuest Central Essentials</collection><collection>ProQuest Central</collection><collection>Technology Collection</collection><collection>Natural Science Collection</collection><collection>ProQuest One Community College</collection><collection>ProQuest Central Korea</collection><collection>Health Research Premium Collection</collection><collection>Health Research Premium Collection (Alumni)</collection><collection>ProQuest Central Student</collection><collection>SciTech Premium Collection</collection><collection>ProQuest Health & Medical Complete (Alumni)</collection><collection>ProQuest Engineering Collection</collection><collection>Agricultural Science Database</collection><collection>Health & Medical Collection (Alumni Edition)</collection><collection>Medical Database</collection><collection>Engineering Database</collection><collection>Environmental Science Database</collection><collection>ProQuest One Academic Eastern Edition (DO NOT USE)</collection><collection>ProQuest One Academic</collection><collection>ProQuest One Academic UKI Edition</collection><collection>ProQuest Central China</collection><collection>Engineering Collection</collection><collection>Environmental Science Collection</collection><collection>SIRS Editorial</collection><collection>MEDLINE - Academic</collection><jtitle>Poultry science</jtitle></facets><delivery><delcategory>Remote Search Resource</delcategory><fulltext>fulltext</fulltext></delivery><addata><au>Julian, R.J</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>Response of the Heart and Pulmonary Arteries to Hypoxia, Pressure, and Volume. A Short Review</atitle><jtitle>Poultry science</jtitle><addtitle>Poult Sci</addtitle><date>2007-05-01</date><risdate>2007</risdate><volume>86</volume><issue>5</issue><spage>1006</spage><epage>1011</epage><pages>1006-1011</pages><issn>0032-5791</issn><eissn>1525-3171</eissn><abstract>The pulmonary arterioles react to hypoxia by contraction and to increased pressure and volume by hypertrophy of the muscular wall, referred to as pulmonary vascular remodeling, both of which increase vascular resistance and result in increased pulmonary arterial pressure. Heart muscle reacts to increased pressure by hypertrophy of cardiac myocytes and thickening of the muscular wall. The heart responds to increased volume by dilation of the chamber that may result in physiologic or pathologic hypertrophy of the muscle wall. Heart muscle cells are very sensitive to hypoxia or other insults, and this may result in death of individual cardiac myocytes with lengthening and thinning of the remaining heart muscle cells and dilation of the chamber in a process called dilated cardiomyopathy.</abstract><cop>England</cop><pub>Oxford University Press</pub><pmid>17435039</pmid><doi>10.1093/ps/86.5.1006</doi><tpages>6</tpages><oa>free_for_read</oa></addata></record> |
| fulltext | fulltext |
| identifier | ISSN: 0032-5791 |
| ispartof | Poultry science, 2007-05, Vol.86 (5), p.1006-1011 |
| issn | 0032-5791 1525-3171 |
| language | eng |
| recordid | cdi_proquest_miscellaneous_70385613 |
| source | MEDLINE; EZB-FREE-00999 freely available EZB journals; Alma/SFX Local Collection |
| subjects | Animals apoptosis blood pressure blood vessels broiler chickens cardiomyopathy dilated cardiomyopathy heart hypertrophy Hypertrophy - pathology Hypertrophy - physiopathology hypoxia Hypoxia - metabolism literature reviews Myocardium - metabolism myocytes Pressure pulmonary arterioles pulmonary artery Pulmonary Artery - anatomy & histology Pulmonary Artery - pathology Pulmonary Artery - physiology Pulmonary Artery - physiopathology vascular resistance vasoconstriction volume |
| title | Response of the Heart and Pulmonary Arteries to Hypoxia, Pressure, and Volume. A Short Review |
| url | https://sfx.bib-bvb.de/sfx_tum?ctx_ver=Z39.88-2004&ctx_enc=info:ofi/enc:UTF-8&ctx_tim=2024-12-26T16%3A14%3A16IST&url_ver=Z39.88-2004&url_ctx_fmt=infofi/fmt:kev:mtx:ctx&rfr_id=info:sid/primo.exlibrisgroup.com:primo3-Article-proquest_cross&rft_val_fmt=info:ofi/fmt:kev:mtx:journal&rft.genre=article&rft.atitle=Response%20of%20the%20Heart%20and%20Pulmonary%20Arteries%20to%20Hypoxia,%20Pressure,%20and%20Volume.%20A%20Short%20Review&rft.jtitle=Poultry%20science&rft.au=Julian,%20R.J&rft.date=2007-05-01&rft.volume=86&rft.issue=5&rft.spage=1006&rft.epage=1011&rft.pages=1006-1011&rft.issn=0032-5791&rft.eissn=1525-3171&rft_id=info:doi/10.1093/ps/86.5.1006&rft_dat=%3Cproquest_cross%3E70385613%3C/proquest_cross%3E%3Curl%3E%3C/url%3E&disable_directlink=true&sfx.directlink=off&sfx.report_link=0&rft_id=info:oai/&rft_pqid=223153422&rft_id=info:pmid/17435039&rfr_iscdi=true |