Regulated and endothelial cell-specific expression of Fas ligand : An in vitro model for a strategy aiming at inhibiting xenograft rejection

Immunologically privileged sites have been shown to express Fas ligand (FasL) and may protect themselves by inducing apoptosis of infiltrating inflammatory cells. We asked whether the Fas/FasL interaction could be used to protect a xenograft from rejection. We proposed that endothelial cells that ar...

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