A possible role for p16INK4 in neuronal cell death after retinal ischemia-reperfusion injury

To study whether cell type-specific death occurs in retinal ischemia-reperfusion injury and the possible roles of p16INK4 in the determination of cell death. Retinal ischemia-reperfusion injury was induced in rats by a ligation method. After 1 hour of ischemia and a time of reperfusion that varied,...

Ausführliche Beschreibung

Gespeichert in:

Bibliographische Detailangaben
Veröffentlicht in:	Investigative ophthalmology & visual science 1999-02, Vol.40 (2), p.528-533
Hauptverfasser:	Kuroiwa, S, Katai, N, Yoshimura, N
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Apoptosis Biological and medical sciences Calbindins Cyclin-Dependent Kinase Inhibitor p16 - physiology Fluorescent Antibody Technique, Indirect In Situ Nick-End Labeling Male Medical sciences Membrane Proteins - metabolism Necrosis Neurons - metabolism Neurons - pathology Neurons - ultrastructure Ophthalmology Qa-SNARE Proteins Rats Rats, Sprague-Dawley Reperfusion Injury - metabolism Reperfusion Injury - pathology Retinal Diseases - metabolism Retinal Diseases - pathology Retinal Vessels - metabolism Retinal Vessels - pathology Retinopathies S100 Calcium Binding Protein G - metabolism
Online-Zugang:	Volltext
Tags:	Tag hinzufügen Keine Tags, Fügen Sie den ersten Tag hinzu!

Schreiben Sie den ersten Kommentar!