Superoxide production, risk factors, and endothelium-dependent relaxations in human internal mammary arteries

In a variety of disease states, endothelium-dependent vasodilation is abnormal. Reduced nitric oxide (NO) production, increased destruction of NO by superoxide, diminished cellular levels of L-arginine or tetrahydrobiopterin, and alterations in membrane signaling have been implicated. We examined th...

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Veröffentlicht in:	Circulation (New York, N.Y.) N.Y.), 1999-01, Vol.99 (1), p.53-59
Hauptverfasser:	HURAUX, C, MAKITA, T, KURZ, S, YAMAGUCHI, K, SZLAM, F, TARPEY, M. M, WILCOX, J. N, HARRISON, D. G, LEVY, J. H
Format:	Artikel
Sprache:	eng
Schlagworte:	Acetylcholine - pharmacology Biological and medical sciences Blood vessels and receptors Calcimycin - pharmacology Endothelium, Vascular - drug effects Endothelium, Vascular - metabolism Female Fundamental and applied biological sciences. Psychology Humans Male Mammary Arteries - drug effects Mammary Arteries - metabolism Middle Aged Nitroglycerin - pharmacology Organ Culture Techniques Risk Factors Superoxides - metabolism Vasodilation - drug effects Vasodilation - physiology Vasodilator Agents - pharmacology Vertebrates: cardiovascular system
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