Suppressive action produced by β-amyloid peptide fragment 31–35 on long-term potentiation in rat hippocampus is N-methyl- d-aspartate receptor-independent: it's offset by (−)huperzine A

Extracellular recordings of field potential from CA1 region of rat hippocampal slices were used to observe the effects of a shorter synthetic fragment of β-amyloid peptide (A β 31–35) on the induction of long-term potentiation (LTP) and the action of (−)huperzine A, a potent acetylcholinesterase (AC...

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