Electric Pulse Stimulation Induces NMDA Glutamate Receptor mRNA in NIH3T3 Mouse Fibroblasts

Excess glutamate and Ca2+ influx into neurons exacerbate brain damage such as ischemia. Astrocytes at the site of damage proliferate and attenuate the glutamate- and Ca2+-induced neuronal damage by removing excess glutamate and Ca2+ through the N-methyl-D-aspartate (NMDA) glutamate receptor and the...

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Veröffentlicht in:	The Tohoku Journal of Experimental Medicine 2008, Vol.215(2), pp.181-187
Hauptverfasser:	Okutsu, Saeko, Hatakeyama, Hiroyasu, Kanazaki, Makoto, Tsubokawa, Hiroshi, Nagatomi, Ryoichi
Format:	Artikel
Sprache:	eng
Schlagworte:	Aniline Compounds - metabolism Animals Ca2+ influx Calcium - metabolism Calcium Channel Blockers - pharmacology Calcium Channels, L-Type - metabolism Cells, Cultured Chelating Agents - pharmacology Egtazic Acid - analogs & derivatives Egtazic Acid - pharmacology electric pulse stimulation (EPS) Electric Stimulation - methods fibroblasts Fibroblasts - metabolism Fluorescent Dyes - metabolism Glutamic Acid - pharmacology Male Mice NIH 3T3 Cells NIH3T3 NMDA receptor Receptors, N-Methyl-D-Aspartate - metabolism RNA, Messenger - metabolism Verapamil - pharmacology Xanthenes - metabolism
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