Beta-catenin triggers nuclear factor kappaB-dependent up-regulation of hepatocyte inducible nitric oxide synthase

Disruption of cell-to-cell contacts, as observed in many pathophysiological conditions, prime hepatocytes for compensatory hyperplastic response that involves induction of several genes, including proto-oncogenes and other gene targets of beta-catenin signaling pathway. By using cultured hepatocytes...

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Veröffentlicht in:	The international journal of biochemistry & cell biology 2008, Vol.40 (9), p.1861-1871
Hauptverfasser:	Bandino, Andrea, Compagnone, Alessandra, Bravoco, Vittoria, Cravanzola, Carlo, Lomartire, Anna, Rossetto, Chiara, Novo, Erica, Cannito, Stefania, Valfrè di Bonzo, Lorenzo, Zamara, Elena, Autelli, Riccardo, Parola, Maurizio, Colombatto, Sebastiano
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals beta Catenin - metabolism beta-Transducin Repeat-Containing Proteins - metabolism Cadherins - metabolism Cattle Cell Communication Cells, Cultured Egtazic Acid - pharmacology Enzyme Induction - drug effects Hepatocytes - enzymology Hepatocytes - metabolism Lithium Chloride - pharmacology Male NF-kappa B - metabolism Nitric Oxide Synthase Type II - biosynthesis Nitric Oxide Synthase Type II - metabolism Protein Transport Rats Reproducibility of Results Up-Regulation - drug effects
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