Whole Body Heat Shock Fails To Protect Mouse Heart Against Ischemia/Reperfusion Injury: Role of 72 kDa Heat Shock Protein and Antioxidant Enzymes

The transgenic mice overexpressing heat shock protein 72 (HSP72) or antioxidants have been reported to be more resistant to myocardial ischemia/reperfusion injury. However, it remains unknown whether whole body heat stress (HS) which may induce HSP72 or endogenous antioxidants affords similar protec...

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Veröffentlicht in:	Journal of molecular and cellular cardiology 1998-11, Vol.30 (11), p.2213-2227
Hauptverfasser:	Xi, Lei, Chelliah, Jeya, Nayeem, Mohammed A., Levasseur, Joseph E., Hess, Michael L., Kukreja, Rakesh C.
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Antioxidants Antioxidants - metabolism Heat shock proteins Heat Stress Disorders Heat-Shock Proteins - metabolism HSP72 Heat-Shock Proteins Ischemia/reperfusion Male Mice Mice, Inbred ICR Mice, Transgenic Mouse Myocardial infarction Myocardial Ischemia - metabolism Myocardial Ischemia - physiopathology Myocardial Ischemia - prevention & control Myocardial Reperfusion Injury - metabolism Myocardial Reperfusion Injury - physiopathology Myocardial Reperfusion Injury - prevention & control Myocardium - metabolism Preconditioning
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